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Systemic venous collateral channel causing desaturation after bidirectional cavopulmonary anastomosis: percutaneous
Satish Karur1, Jayaranganath Mahima, Manjunath Cholenahally Nanjappa
1Department of Cardiology, Sri Jayadeva Institute of Cardiovascular Sciences and Research, Bangalore, India. drsatishkdm@yahoo.com
A large venous collateral caused worsening cyanosis in a child 5 years after bidirectional Glenn shunt surgery. Percutaneous closure of this abnormal vessel successfully restored oxygen levels, highlighting an effective treatment for this complication.
Area of Science:
- Pediatric Cardiology
- Interventional Cardiology
- Congenital Heart Disease
Background:
- Cyanotic cardiac disease requires surgical palliation, such as the bidirectional Glenn shunt.
- Post-surgical complications can arise, impacting long-term outcomes.
- Progressive cyanosis necessitates investigation into shunt function and potential obstructions.
Observation:
- A patient with a history of bidirectional Glenn shunt developed worsening cyanosis 5 years post-surgery.
- Imaging revealed an unusually large venous collateral decompressing the bidirectional Glenn shunt.
- This collateral provided an abnormal pathway from the superior vena cava to the inferior vena cava.
Findings:
- The large venous collateral was identified as the cause of the patient's recurrent cyanosis.
- Percutaneous closure of the venous collateral using a vascular plug was performed.
- The procedure resulted in excellent clinical outcomes and resolution of cyanosis.
Implications:
- Unusual venous collaterals can develop after bidirectional Glenn shunt procedures.
- Percutaneous closure is a viable and effective treatment for such complex venous anomalies.
- This approach offers a minimally invasive solution for managing post-surgical complications in pediatric cardiac patients.
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