Identification and evaluation of constitutively active thyroid stimulating hormone receptor mutations

Joaquin Lado-Abeal1, Leah R Quisenberry, Isabel Castro-Piedras

  • 1Department of Internal Medicine, Tech University Health Sciences Center-SOM, Lubbock, Texas, USA.

Methods in Enzymology
|November 2, 2010
PubMed

Insights

Constitutive mutations in the thyroid stimulating hormone receptor (TSHR) gene can cause thyroid dysfunction. This study details cost-effective methods for analyzing these TSHR mutations and their functional impacts.

Area of Science:

  • Endocrinology
  • Molecular Biology
  • Genetics

Background:

  • The thyroid stimulating hormone receptor (TSHR) is a G protein-coupled receptor regulating thyroid gland function.
  • TSHR activation is typically mediated by thyroid stimulating hormone (TSH); however, mutations can lead to constitutive activity.
  • Constitutively active TSHR mutations are frequently found in the serpentine region, particularly the third intracellular loop or transmembrane domain six.

Purpose of the Study:

  • To describe simple and cost-effective laboratory methods for studying TSHR constitutive mutations.
  • To investigate the molecular mechanisms and functional consequences of TSHR activating mutations.

Main Methods:

  • Screening TSHR gene mutations in paraffin-embedded thyroid tissue.
  • In vitro measurement of TSHR constitutive activity.
  • Flow cytometry analysis for cell surface TSHR expression.
  • Analysis of TSH binding to TSHR.
  • TSHR phosphorylation assays.

Main Results:

  • The study presents a suite of accessible methods for TSHR mutation analysis.
  • These methods allow for the comprehensive study of TSHR constitutive activity, expression, ligand binding, and phosphorylation.

Conclusions:

  • The described methods provide a practical approach to investigating the genetic and functional aspects of TSHR mutations.
  • These techniques facilitate the study of thyroid diseases associated with constitutively active TSHR.

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