Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Encephalitis ll: Pathophysiology01:26

Encephalitis ll: Pathophysiology

Encephalitis is inflammation of the brain parenchyma caused by direct viral invasion or immune-mediated mechanisms triggered by infections or tumors. Both processes lead to neuronal injury, disrupted neurotransmission, and diverse neurological symptoms, often with overlapping clinical and pathological features.Autoimmune EncephalitisIn autoimmune encephalitis, antibodies target neuronal antigens on cell surfaces, synapses, or within neurons. A key example is anti-NMDAR encephalitis, which can...
Arboviral Encephalitis01:25

Arboviral Encephalitis

Arboviral encephalitis refers to brain inflammation caused by arthropod-borne viruses, particularly those transmitted through mosquito vectors. Among these, West Nile virus (WNV), a member of the Flaviviridae family, is a significant public health concern. WNV is an enveloped, positive-sense, single-stranded RNA virus. Human infection typically begins when an infected mosquito introduces the virus into the dermis during feeding. The primary transmission cycle involves birds as amplifying hosts...
Encephalitis l: Introduction01:19

Encephalitis l: Introduction

Encephalitis is inflammation of the brain parenchyma, most often due to infections or autoimmune processes. It presents with neuropsychiatric features such as fever, altered mental status, behavioral changes, cognitive dysfunction, seizures, focal deficits, and sometimes autonomic instability. In some cases, the meninges are also involved, resulting in meningoencephalitis.Infectious CausesInfectious encephalitis is most commonly viral but can also result from bacterial, fungal, or parasitic...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

<i>In Vivo</i> confocal microscopy evaluation of corneal layer alterations in patients with trigeminal neuralgia.

Cephalalgia : an international journal of headache·2026
Same author

Melatonin and oxidative DNA damage in REM- and NREM-related obstructive sleep apnea: an exploratory analysis of sleep-stage phenotypes.

European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery·2026
Same author

Transcranial doppler-derived cerebrovascular resistance changes during vagus nerve stimulation in drug-resistant epilepsy.

Epilepsy research·2026
Same author

Raeder's paratrigeminal neuralgia secondary to internal carotid artery dissection mimicking trigeminal autonomic cephalalgia: a case report.

Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology·2026
Same author

Automated scoring of student videos in medical education: a comparison between a large language model and expert evaluation.

Journal of microbiology & biology education·2026
Same author

An evaluation of flipped classroom-based anatomy teaching in medical students.

Irish journal of medical science·2026

Related Experiment Video

Updated: Jun 6, 2026

Visual Evoked Potential Recording in a Rat Model of Experimental Optic Nerve Demyelination
06:49

Visual Evoked Potential Recording in a Rat Model of Experimental Optic Nerve Demyelination

Published on: July 29, 2015

Visual evoked potentials in neurobrucellosis.

Ufuk Ergun1, Gunay T Ertem, Gulnihal Kutlu

  • 1Department of Neurology, Ministry of Health, Ankara Education and Research Hospital, Ankara, Turkey.

Neurosciences (Riyadh, Saudi Arabia)
|November 11, 2010
PubMed
Summary
This summary is machine-generated.

Visual evoked potentials (VEPs) can help differentiate neurobrucellosis from brucellosis. Lower P100 amplitude in VEPs indicates axonal damage in neurobrucellosis patients.

More Related Videos

In Vivo Methods to Assess Retinal Ganglion Cell and Optic Nerve Function and Structure in Large Animals
12:18

In Vivo Methods to Assess Retinal Ganglion Cell and Optic Nerve Function and Structure in Large Animals

Published on: February 26, 2022

How to Obtain Reliable Visual Event-related Potentials in Newborns
07:39

How to Obtain Reliable Visual Event-related Potentials in Newborns

Published on: October 24, 2019

Related Experiment Videos

Last Updated: Jun 6, 2026

Visual Evoked Potential Recording in a Rat Model of Experimental Optic Nerve Demyelination
06:49

Visual Evoked Potential Recording in a Rat Model of Experimental Optic Nerve Demyelination

Published on: July 29, 2015

In Vivo Methods to Assess Retinal Ganglion Cell and Optic Nerve Function and Structure in Large Animals
12:18

In Vivo Methods to Assess Retinal Ganglion Cell and Optic Nerve Function and Structure in Large Animals

Published on: February 26, 2022

How to Obtain Reliable Visual Event-related Potentials in Newborns
07:39

How to Obtain Reliable Visual Event-related Potentials in Newborns

Published on: October 24, 2019

Area of Science:

  • Ophthalmology
  • Neurology
  • Infectious Diseases

Background:

  • Brucellosis is a zoonotic infection with potential neurological complications.
  • Neurobrucellosis presents diagnostic challenges, necessitating sensitive detection methods.

Purpose of the Study:

  • To evaluate the utility of visual evoked potentials (VEPs) in distinguishing brucellosis from neurobrucellosis.
  • To assess VEP P100 latency and amplitude differences between patient groups.

Main Methods:

  • 23 patients diagnosed with brucellosis were included.
  • Visual evoked potentials (VEPs) were recorded after neurological and ophthalmological examinations.
  • P100 latency and amplitude were compared between brucellosis (n=17) and neurobrucellosis (n=6) groups.

Main Results:

  • No significant difference in mean P100 latency was observed between brucellosis and neurobrucellosis groups (p=0.38).
  • Neurobrucellosis patients exhibited significantly lower mean P100 amplitude compared to brucellosis patients (p=0.012).

Conclusions:

  • VEP P100 amplitude reduction may serve as a distinctive indicator of axonal pathology in neurobrucellosis.
  • VEPs show potential as a diagnostic tool for identifying neurological involvement in brucellosis.