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Type I Diabetes II: Pathophysiology01:26

Type I Diabetes II: Pathophysiology

Type 1 diabetes mellitus arises from an immune-mediated destruction of pancreatic β-cells, resulting in an absolute deficiency of insulin. This process develops in genetically susceptible individuals when autoimmunity, environmental exposures, and immunologic dysregulation converge to trigger a targeted attack on the insulin-producing cells of the pancreas. The β-cells are located within the islets of Langerhans and are essential for regulating blood glucose by facilitating cellular uptake of...

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Tumor Engraftment in a Xenograft Mouse Model of Human Mantle Cell Lymphoma
10:52

Tumor Engraftment in a Xenograft Mouse Model of Human Mantle Cell Lymphoma

Published on: March 30, 2018

Subcutaneous panniculitis-like T-cell lymphoma.

Abel Francis1, S Criton, Sandhya Acharya

  • 1Assistant Professor, Department of Dermatology, Amala Institute of Medical Sciences, (AIMS), Thrissur, Kerala, India.

Indian Journal of Dermatology
|November 11, 2010
PubMed
Summary
This summary is machine-generated.

This case report details a subcutaneous panniculitis-like T-cell lymphoma (SPTCL) diagnosis in a 38-year-old woman. Immunohistochemical findings indicated an indolent tumor course, managed successfully with corticosteroids.

Keywords:
Subcutaneous panniculitis-likeT-cell lymphomarimming of fat cells

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Area of Science:

  • Dermatology
  • Hematology
  • Oncology

Background:

  • Subcutaneous panniculitis-like T-cell lymphoma (SPTCL) is a rare form of cutaneous T-cell lymphoma.
  • Accurate diagnosis relies on integrating clinical, histopathological, and immunohistochemical findings.

Purpose of the Study:

  • To present a case report of SPTCL.
  • To highlight the diagnostic utility of immunohistochemistry (IHC) in characterizing SPTCL.
  • To discuss the management and clinical course of a patient with SPTCL.

Main Methods:

  • Clinical presentation review.
  • Histopathological examination of skin biopsy.
  • Immunohistochemical (IHC) analysis including CD8 and CD56 markers.

Main Results:

  • The patient presented with clinical and histopathological features consistent with SPTCL.
  • IHC revealed CD8-positive and CD56-negative T-cells, suggesting an indolent tumor behavior.
  • The patient showed good clinical improvement with corticosteroid therapy.

Conclusions:

  • SPTCL diagnosis requires a multidisciplinary approach.
  • Specific IHC markers (CD8+, CD56-) can predict an indolent course in SPTCL.
  • Corticosteroids can be an effective treatment modality for SPTCL.