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Updated: Jun 5, 2026

Laparoscopic Left Hemihepatectomy Combined with Caudate Lobe Resection
Published on: April 11, 2025
Huge caudate lobe of the liver due to Budd-Chiari syndrome
Yuta Kikuchi1, Hiroshi Yoshida, Yasuhiro Mamada
1Surgery for Organ Function and Biological Regulation, Graduate School of Medicine, Nippon Medical School, Tokyo, Japan.
Insights
Budd-Chiari syndrome can cause an enlarged liver caudate lobe. This case study details a patient treated for gastric varices, highlighting successful management of hepatic venous outflow obstruction.
Area of Science:
- Hepatology
- Vascular Surgery
- Gastroenterology
Background:
- Budd-Chiari syndrome involves hepatic venous outflow obstruction.
- Idiopathic portal hypertension can lead to liver deformities and varices.
- Enlarged gastric varices necessitate treatment to prevent complications.
Observation:
- A patient presented with solitary gastric varices and a significantly enlarged liver caudate lobe.
- Imaging revealed hepatic vein obstructions and scars, characteristic of Budd-Chiari syndrome.
- The caudate lobe's drainage vein was dilated, while other hepatic veins showed no atrophy.
Findings:
- The diagnosis was Budd-Chiari syndrome with solitary gastric varices.
- The enlarged caudate lobe was a key feature associated with the hepatic venous outflow obstruction.
- No inferior vena cava obstruction was detected, differentiating from other venous conditions.
Implications:
- This case demonstrates a rare presentation of Budd-Chiari syndrome with caudate lobe enlargement.
- Successful treatment of gastric varices was achieved using balloon-occluded retrograde transvenous obliteration and splenic embolization.
- The findings underscore the importance of comprehensive imaging in diagnosing complex hepatic vascular conditions.
Abstract:
Budd-Chiari syndrome is characterized by hepatic venous outflow obstruction. We describe a patient with a huge caudate lobe of the liver due to Budd-Chiari syndrome. A 49-year-old woman was referred to Nippon Medical School Hospital to receive treatment for enlarged gastric varices. She had been followed up for idiopathic portal hypertension with deformity of the liver for 7 years and had undergone surgery for pharyngeal carcinoma 5 years earlier. Upper gastrointestinal endoscopy revealed solitary gastric varices without esophageal varices. Abdominal computed tomography revealed obstructions and scars of the right, middle, and left hepatic veins. The caudate lobe was enlarged, and the portal vein was displaced ventrally, without thrombus. The drainage vein of the caudate lobe, the short hepatic vein on the left side of the inferior vena cava, was dilated. The portal vein and drainage vein of segment 6 were visualized and showed no atrophy. Venography revealed no obstruction of the inferior vena cava. Budd-Chiari syndrome with solitary gastric varices was diagnosed on the basis of these findings. We performed balloon-occluded retrograde transvenous obliteration and partial splenic embolization to treat the gastric varices. The posttreatment course was uneventful, and the patient was discharged 8 days after embolization. The gastric varices shrank.
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