Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Genomic Imprinting and Inheritance02:30

Genomic Imprinting and Inheritance

Diploid organisms inherit genetic material through chromosomes from both parents. Copies of the same gene are known as alleles. In most cases, both alleles are simultaneously expressed and allow various cellular processes to function optimally. If one of the alleles is missing or mutated, the expression of the other allele can compensate; however, this is not true for all genes.
The expression of some genes depends on which parent passed the gene to the offspring, through a phenomenon known as...
Pedigree Analysis01:35

Pedigree Analysis

Overview
Pedigree Analysis01:35

Pedigree Analysis

Overview
Sex-linked Disorders01:43

Sex-linked Disorders

Like autosomes, sex chromosomes contain a variety of genes necessary for normal body function. When a mutation in one of these genes results in biological deficits, the disorder is considered sex-linked.
Law of Segregation01:49

Law of Segregation

When crossing pea plants, Mendel noticed that one of the parental traits would sometimes disappear in the first generation of offspring, called the F1 generation, and could reappear in the next generation (F2). He concluded that one of the traits must be dominant over the other, thereby causing masking of one trait in the F1 generation. When he crossed the F1 plants, he found that 75% of the offspring in the F2 generation had the dominant phenotype, while 25% had the recessive phenotype.
Nondisjunction01:21

Nondisjunction

Nondisjunction is the failure of homologous chromosomes or sister chromatids to separate correctly and move to the opposite poles of the cells. This produces daughter cells with abnormal chromosome numbers.  Nondisjunction is common during anaphase I or anaphase II of meiosis.  Mutations in synaptonemal complex proteins that attach homologous chromosomes increase the chances of nondisjunction in anaphase I of meiosis I. In contrast, mutations in topoisomerases and condensins that hold sister...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Golf and Binocular Vision.

The Hospital·2018
Same author

Treatment of Pulmonary Phthisis.

The Hospital·2018
Same author

SOME COMPARISON OF PHTHISICAL WITH NON-TUBERCULOUS MALES, CHIEFLY IN RESPECT OF HEALTHY HEIGHT-WEIGHT RATIO, AND OF MOUTH BREATHING.

British medical journal·2010
Same author

THE COMPARATIVE FREQUENCY OF IMPAIRED NASAL RESPIRATION AS AN ANTECEDENT TO PULMONARY AND TO EXTRAPULMONARY TUBERCULOSIS.

British medical journal·2010
Same author

THE DROOPING SHOULDER SIGN OF PHTHISIS.

British medical journal·2010
Same journal

Social and genetic influences on life and death : A review.

The Eugenics review·2011
Same journal

Genetic and environmental factors in human ability.

The Eugenics review·2011
Same journal

The role of genetic factors in the human face, jaws and teeth: a review.

The Eugenics review·2011
Same journal

Sociology, biology and population control.

The Eugenics review·2011
Same journal

Your heredity and environment.

The Eugenics review·2011
Same journal

Fertility differentials in England and Wales: Some facts.

The Eugenics review·2011
See all related articles

Related Experiment Video

Updated: Jun 5, 2026

A Strategy to Identify de Novo Mutations in Common Disorders such as Autism and Schizophrenia
05:51

A Strategy to Identify de Novo Mutations in Common Disorders such as Autism and Schizophrenia

Published on: June 15, 2011

Primogeniture and abnormality

W C Rivers

    The Eugenics Review
    |January 25, 2011
    PubMed
    Summary

    No abstract available in PubMed .

    More Related Videos

    A Novel Strategy Combining Array-CGH, Whole-exome Sequencing and In Utero Electroporation in Rodents to Identify Causative Genes for Brain Malformations
    08:22

    A Novel Strategy Combining Array-CGH, Whole-exome Sequencing and In Utero Electroporation in Rodents to Identify Causative Genes for Brain Malformations

    Published on: December 1, 2017

    Microsatellite DNA Genotyping and Flow Cytometry Ploidy Analyses of Formalin-fixed Paraffin-embedded Hydatidiform Molar Tissues
    11:54

    Microsatellite DNA Genotyping and Flow Cytometry Ploidy Analyses of Formalin-fixed Paraffin-embedded Hydatidiform Molar Tissues

    Published on: October 20, 2019

    Related Experiment Videos

    Last Updated: Jun 5, 2026

    A Strategy to Identify de Novo Mutations in Common Disorders such as Autism and Schizophrenia
    05:51

    A Strategy to Identify de Novo Mutations in Common Disorders such as Autism and Schizophrenia

    Published on: June 15, 2011

    A Novel Strategy Combining Array-CGH, Whole-exome Sequencing and In Utero Electroporation in Rodents to Identify Causative Genes for Brain Malformations
    08:22

    A Novel Strategy Combining Array-CGH, Whole-exome Sequencing and In Utero Electroporation in Rodents to Identify Causative Genes for Brain Malformations

    Published on: December 1, 2017

    Microsatellite DNA Genotyping and Flow Cytometry Ploidy Analyses of Formalin-fixed Paraffin-embedded Hydatidiform Molar Tissues
    11:54

    Microsatellite DNA Genotyping and Flow Cytometry Ploidy Analyses of Formalin-fixed Paraffin-embedded Hydatidiform Molar Tissues

    Published on: October 20, 2019