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Related Concept Videos

Spinal Nerves: Plexus I01:22

Spinal Nerves: Plexus I

Nerve plexuses are networks of interlacing nerves that serve as communication hubs to distribute and organize nerve action across various body regions. The nerve plexuses are organized into the cervical plexus located in the neck region, brachial plexus in the shoulder area, lumbar plexus found in the lower back, sacral plexus situated in the pelvis, and coccygeal plexus located in the coccygeal region.
The Cervical Plexus
The cervical plexus, formed by the anterior rami of the first four...
Spinal Nerves: Plexus II01:21

Spinal Nerves: Plexus II

The plexuses of the lower body include the lumbar, sacral, and coccygeal plexuses, which innervate the abdomen, pelvis, legs, and coccygeal region. These plexuses control the transmission of sensory information and coordinate motor functions of the lower body.
The Lumbar Plexus
The lumbar plexus is situated within the lumbar region of the back and is primarily formed by the first four lumbar spinal nerves (L1 to L4). This plexus extends its branches into several nerves, including the...
Spinal Cord Injury ll: Pathophysiology01:14

Spinal Cord Injury ll: Pathophysiology

Spinal cord injury progresses through two interconnected phases: primary injury and secondary injury.Primary InjuryPrimary injury happens at the moment of trauma and involves immediate mechanical damage to the spinal cord.Compression happens when broken vertebrae, herniated discs, or accumulating blood (such as a hematoma) press directly against the spinal cord, distorting its normal shape and function. In cases of contusion, the cord is bruised by a blunt force (like penetrating injuries or...
Secondary Spinal Cord Injury llI: Pathophysiology01:25

Secondary Spinal Cord Injury llI: Pathophysiology

Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...

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Related Experiment Video

Updated: Jun 3, 2026

Development of a Neonatal Rat Model for Brachial Plexus Birth Injury
09:42

Development of a Neonatal Rat Model for Brachial Plexus Birth Injury

Published on: March 27, 2026

22. Traumatic plexus lesion.

Robert van Dongen1, Steven P Cohen, Maarten van Kleef

  • 1Department of Anesthesiology Pain and Palliative Medicine, Radboud University Nijmegen Medical Centre, Nijmegen, The Netherlands.

Pain Practice : the Official Journal of World Institute of Pain
|March 31, 2011
PubMed
Summary
This summary is machine-generated.

Traumatic brachial plexus injuries cause pain and deficits, necessitating early physical therapy and potential surgical repair. Neurostimulation may offer additional benefits for managing complex neuropathic pain in these patients.

More Related Videos

Structured Motor Rehabilitation After Selective Nerve Transfers
09:34

Structured Motor Rehabilitation After Selective Nerve Transfers

Published on: August 15, 2019

Related Experiment Videos

Last Updated: Jun 3, 2026

Development of a Neonatal Rat Model for Brachial Plexus Birth Injury
09:42

Development of a Neonatal Rat Model for Brachial Plexus Birth Injury

Published on: March 27, 2026

Structured Motor Rehabilitation After Selective Nerve Transfers
09:34

Structured Motor Rehabilitation After Selective Nerve Transfers

Published on: August 15, 2019

Area of Science:

  • Neurology
  • Neurosurgery
  • Physical Therapy

Background:

  • Traumatic brachial plexus injuries lead to significant pain, motor, and sensory deficits.
  • Co-existing severe injuries often require prompt surgical intervention.
  • Effective management strategies are crucial for patient recovery.

Purpose of the Study:

  • To review the current understanding of managing traumatic brachial plexus injuries.
  • To highlight the importance of early rehabilitation and physical therapy.
  • To explore the potential role of neurostimulation in managing neuropathic pain.

Main Methods:

  • Review of existing literature on brachial plexus injury management.
  • Analysis of treatment outcomes from historical reports.
  • Discussion of pharmacological, surgical, and neurostimulation approaches.

Main Results:

  • Treatment results for brachial plexus injuries vary significantly, often based on small historical studies.
  • Early rehabilitation and physical therapy are established as essential components of care.
  • Surgical reconstruction is a common approach for nerve disruption.

Conclusions:

  • Management of traumatic brachial plexus injuries requires a multidisciplinary approach.
  • Neurostimulation presents a potential adjunctive therapy for neuropathic pain in complex cases.
  • Further research is needed to optimize outcomes for these challenging injuries.