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5-HT1A, 5-HT1B and 5-HT2 receptor agonists induce differential behavioral responses in preweanling rat pups

N A Frambes1, C L Kirstein, C A Moody

  • 1Department of Psychology, Suny-Binghamton 13901.

Insights

Serotonin agonists 8-OH-DPAT, ipsapirone, mCPP, and DOI affect behaviors in preweanling rats differently than in neonates. These developmental changes in serotonin receptor function may influence early life behaviors.

Area of Science:

  • Neuroscience
  • Developmental Biology
  • Pharmacology

Background:

  • Serotonergic systems play a crucial role in neurodevelopment.
  • Understanding the ontogeny of serotonin receptor function is vital for comprehending behavioral development.

Purpose of the Study:

  • To investigate the effects of specific serotonin receptor agonists on behaviors in preweanling rats.
  • To compare these effects with those observed in neonatal rats to identify developmental changes.

Main Methods:

  • Preweanling Sprague-Dawley rats (postnatal day 17-18) were administered various doses of 5-HT1A agonists (8-OH-DPAT, ipsapirone), a 5-HT1B agonist (mCPP), and a 5-HT2 agonist (DOI).
  • Behaviors including mouthing, grooming, and limb positioning were assessed in the presence and absence of milk.
  • Responses were compared to previously reported data from neonatal rats.

Main Results:

  • 8-OH-DPAT decreased mouthing behavior, while ipsapirone, mCPP, and DOI had no effect.
  • All four agonists significantly reduced grooming behavior.
  • 8-OH-DPAT and mCPP altered limb positioning, causing hindlimb control issues and a hindlimb straddle, respectively.
  • Functional responses to these agonists differed between preweanling and neonatal rats.

Conclusions:

  • Serotonin receptor-mediated behaviors exhibit significant ontogenetic changes from the neonatal to preweanling period.
  • These developmental alterations in serotonergic function may underlie observed changes in behaviors like mouthing and suckling.
  • The study highlights the dynamic nature of the developing brain's response to neurotransmitter systems.

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