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Published on: August 8, 2017
Selenium toxicosis in a white-tailed deer herd.
Ahmad N Al-Dissi1, Barry R Blakley, Murray R Woodbury
1Department of Veterinary Biomedical Sciences, Western College of Veterinary Medicine, University of Saskatchewan, 52 Campus Drive, Saskatoon, Saskatchewan S7N 5B4. ahmad.aldissi@usask.ca
Chronic selenium toxicosis caused anorexia, weight loss, and lameness in white-tailed deer. Liver selenium levels were elevated, with myocardial damage observed, marking the first report in this species.
Area of Science:
- Veterinary Toxicology
- Wildlife Health
- Environmental Contaminants
Background:
- Selenium toxicosis is a significant concern in wildlife populations.
- Understanding the effects of environmental contaminants on free-ranging animals is crucial for conservation.
Observation:
- A herd of white-tailed deer exhibited clinical signs including anorexia, weight loss, and lameness.
- Elevated selenium concentrations (2.7–8.97 mg/kg wet weight) were detected in the liver tissues of affected deer.
Findings:
- Histopathological examination revealed myocardial necrosis, mineralization, and fibroplasia in the deer.
- This study documents the first confirmed case of chronic selenium toxicosis in white-tailed deer.
Implications:
- This finding highlights the susceptibility of white-tailed deer to selenium poisoning.
- Environmental monitoring for selenium is recommended in areas inhabited by white-tailed deer to prevent further toxicosis events.
- This research contributes to the understanding of selenium's impact on cervid health and informs wildlife management strategies.
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