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[Intussusception after congenital diaphragmatic hernia repair]
A Mazzei1, E Baldassarre, A Centonze
1Service de chirurgie pédiatrique, hôpital Pugliese-Ciaccio, Catanzaro, Italie.
Insights
Intussusception after congenital diaphragmatic hernia (CDH) repair is rare. This case report suggests postoperative bowel atony may cause intussusception by acting as a functional leading point.
Area of Science:
- Pediatric Surgery
- Gastrointestinal Surgery
Background:
- Bowel intussusception is a known complication following abdominal surgery.
- Literature specifically addressing intussusception after congenital diaphragmatic hernia (CDH) repair is limited.
Observation:
- A 24-month-old female presented with symptoms of intestinal obstruction post-CDH repair.
- Exploratory laparotomy revealed an ileoileal intussusception without an identifiable leading point.
Findings:
- The intussusception occurred 15 cm from the ileocecal valve.
- The patient experienced worsening symptoms on postoperative day 7.
Implications:
- Postoperative bowel atony in the herniated segment may act as a functional leading point, triggering intussusception.
- This highlights a potential, albeit uncommon, complication in pediatric CDH surgical management.
Introduction:
Bowel intussusception is a common complication of abdominal surgery. However, the literature on intussusception after congenital diaphragmatic hernia (CDH) repair is scarce.
Case Report:
A 24-month-old female was admitted with vomiting, crying and leukocytosis, with no objective abdominal signs. The chest x-ray showed the presence of bowel in the left hemithorax. Surgical exposure reduced a hernia across a Bochdalek defect, involving part of the left colon and the transverse colon. On the 7th postoperative day, the patient had symptoms of intestinal obstruction with worsening of her general condition. The explorative laparotomy evidenced an ileoileal intussusception, 15 cm from the ileocecal valve, in absence of a leading point.
Conclusion:
A postoperative intussusception in a similar case could be explained by atony of the herniated bowel, possibly a functional leading point in the postoperative phase, when the peristalsis is reactivated.
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