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Related Concept Videos

Pulmonary Hypertension: Classification and Pathogenesis01:30

Pulmonary Hypertension: Classification and Pathogenesis

Pulmonary hypertension (PH) is a severe health condition in which the mean pulmonary arterial pressure increases to 25 mmHg or more, even when the body is at rest. This high pressure in the blood vessels that transport blood from the heart to the lungs can cause various symptoms, including shortness of breath, can lead to right heart failure, and significantly affect the overall quality of life.
There are various classifications for PH, each relating to different underlying causes and also...
Pulmonary Edema II: Pathophysiology01:18

Pulmonary Edema II: Pathophysiology

Pulmonary edema is the accumulation of fluid in the interstitial and alveolar spaces of the lungs, impairing gas exchange and oxygen delivery. It may be cardiogenic or noncardiogenic, but both reduce oxygenation and lung compliance.Cardiogenic Pulmonary EdemaCardiogenic edema results from increased hydrostatic pressure in pulmonary capillaries, usually due to left ventricular dysfunction from myocardial infarction, heart failure, or valvular disease. Ineffective cardiac pumping causes blood to...
Pneumothorax II: Pathophysiology01:08

Pneumothorax II: Pathophysiology

Pneumothorax means the presence of air in the pleural space — the thin potential gap between the visceral and parietal pleura. This condition disrupts the normal pressure balance that keeps the lungs inflated, leading to partial or complete collapse of the affected lung.Normal physiologyUnder normal conditions, the pleural space maintains a slightly negative intrapleural pressure, which keeps the lungs expanded against the chest wall. This negative pressure creates a delicate balance between...
Pulmonary Tuberculosis II01:28

Pulmonary Tuberculosis II

Tuberculosis, or TB, is a bacterial infectious disease caused by Mycobacterium tuberculosis. While its primary impact is on the lungs, leading to pulmonary tuberculosis, it can also affect various other organs, a condition referred to as extrapulmonary tuberculosis.
Here is a detailed explanation of its pathophysiology:
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Pulmonary Embolism II: Diagnostic Studies and Interprofessional Care01:29

Pulmonary Embolism II: Diagnostic Studies and Interprofessional Care

Diagnosing Pulmonary EmbolismDiagnosing pulmonary embolism (PE) involves clinical assessment and advanced imaging tests. The preferred diagnostic tool is the spiral (helical) CT scan or CT angiography (CTA), which uses intravenous contrast media to visualize the pulmonary vasculature and identify emboli.A ventilation-perfusion (V/Q) scan is an alternative for patients unable to receive contrast media. This scan includes both perfusion and ventilation scanning. Perfusion scanning involves...
Other Pulmonary Disorders01:17

Other Pulmonary Disorders

Respiratory disorders encompass a range of conditions with varying levels of severity. Asthma, marked by chronic airway inflammation and hypersensitivity, is one such condition. It can lead to airway obstruction due to factors like bronchial spasms, mucosal edema, increased mucus secretion, or epithelial damage. Asthma triggers are diverse, ranging from allergens to emotional upset, and treatment focuses on both immediate relief through bronchodilators and long-term inflammation suppression.

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Updated: Jun 2, 2026

Left Atrial Stenosis Induced Pulmonary Venous Arterialization and Group 2 Pulmonary Hypertension in Rat
08:34

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Published on: November 18, 2018

[Pulmonary arteriovenous malformation: two sibling cases].

Bülent Mustafa Yenigün1, Cabir Yüksel, Serkan Enön

  • 1Department of Chest Surgery, Faculty of Medicine, Ankara University, Ankara, Turkey. drbulent18@hotmail.com

Tuberkuloz Ve Toraks
|May 11, 2011
PubMed
Summary

Multiple pulmonary arteriovenous malformations can be associated with hereditary hemorrhagic telangiectasia (HHT). Familial screening identified HHT in a patient

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Area of Science:

  • Vascular Medicine
  • Genetics
  • Radiology

Background:

  • Pulmonary arteriovenous malformations (PAVMs) represent abnormal vascular connections within the lungs.
  • PAVMs can occur as isolated anomalies or as multiple lesions.

Observation:

  • This case report details two patients diagnosed with multiple PAVMs.
  • Familial screening was conducted due to the possibility of autosomal dominant hereditary hemorrhagic telangiectasia (HHT), also known as Rendu-Osler-Weber Syndrome (ROWS).

Findings:

  • Multiple PAVMs were identified in both patients.
  • A sister of one patient was also diagnosed with PAVMs, suggesting a familial link.

Implications:

  • Highlights the importance of considering HHT in patients with multiple PAVMs.
  • Emphasizes the utility of familial screening for early diagnosis and management of HHT and associated PAVMs.