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Ethanol and neuronal calcium channels.

S W Leslie1, L M Brown, J E Dildy

  • 1Division of Pharmacology, College of Pharmacy, University of Texas at Austin 78712.

Alcohol (Fayetteville, N.Y.)
|May 1, 1990
PubMed
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Ethanol affects calcium channels, including those linked to N-methyl-D-aspartate (NMDA) receptors. This review explores the physiological significance of these ethanol-induced calcium channel alterations and their link to behavioral effects.

Area of Science:

  • Neuroscience
  • Pharmacology

Background:

  • Ethanol is known to interact with cellular calcium transport mechanisms.
  • Voltage-dependent and receptor-operated calcium channels are potential targets of ethanol.

Purpose of the Study:

  • To review the effects of ethanol on calcium-related parameters.
  • To investigate the physiological significance of ethanol's impact on calcium channels.
  • To correlate these changes with ethanol's behavioral effects.

Main Methods:

  • Literature review of studies on ethanol and calcium channels.
  • Analysis of research on calcium homeostasis and ethanol.
  • Synthesis of findings linking calcium channel modulation to behavior.

Main Results:

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  • Ethanol modifies the function of voltage-dependent calcium channels.
  • Ethanol also affects receptor-operated calcium channels, such as those involving N-methyl-D-aspartate (NMDA) receptors.
  • The physiological relevance of these alterations is still under investigation.

Conclusions:

  • Ethanol's impact on calcium channels is multifaceted.
  • Understanding these effects is crucial for explaining ethanol's behavioral consequences.
  • Further research is needed to establish definitive physiological links.