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Influence of ciprofloxacin on leukotriene generation from various cells in vitro

J Knöller1, J Brom, W Schönfeld

  • 1Lehrstuhl für Medizinische Mikrobiologie und Immunologie, Ruhr Universität Bochum, FRG.

Insights

Ciprofloxacin significantly suppresses leukotriene generation in human immune cells. However, it increases LTB4-receptor expression on polymorphonuclear leukocytes (PMNs) and affects 12-HETE synthesis in platelets.

Area of Science:

  • Immunopharmacology
  • Cellular Biology

Background:

  • Leukotrienes play crucial roles in inflammatory responses.
  • Polymorphonuclear leukocytes (PMNs) and other immune cells generate leukotrienes.
  • Ciprofloxacin is a widely used antibiotic with potential immunomodulatory effects.

Purpose of the Study:

  • To investigate the impact of ciprofloxacin on leukotriene generation from human PMNs and lymphocyte, monocyte, and basophil (LMB) fractions.
  • To examine ciprofloxacin's influence on LTB4-receptor expression in PMNs.
  • To analyze the effect of ciprofloxacin on 12-hydroxyeicosatetraenoic acid (12-HETE) synthesis in human platelets.

Main Methods:

  • Human PMNs and LMB fractions were preincubated with varying concentrations of ciprofloxacin.
  • Leukotriene generation was induced using the calcium-ionophore A23187.
  • Analysis of leukotrienes was performed using high-performance liquid chromatography (HPLC).
  • LTB4-receptor expression on PMNs and 12-HETE synthesis in platelets were also analyzed.

Main Results:

  • Ciprofloxacin significantly suppressed calcium-ionophore-induced leukotriene generation from PMNs and LMBs in a dose-dependent manner.
  • Incubation with ciprofloxacin led to an augmentation of LTB4-receptor expression on PMNs.
  • Ciprofloxacin exhibited dual effects on 12-HETE synthesis in platelets: suppression at high concentrations and increased synthesis at lower concentrations.

Conclusions:

  • Ciprofloxacin demonstrates immunomodulatory effects by suppressing leukotriene generation.
  • The antibiotic influences inflammatory mediator pathways, including LTB4-receptor expression and 12-HETE synthesis.
  • These findings suggest complex interactions between ciprofloxacin and human immune cell functions relevant to inflammation.

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