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Direct activation of cdc2 with phosphatase: identification of p13suc1-sensitive and insensitive steps

C Jessus1, B Ducommun, D Beach

  • 1Cold Spring Harbor Laboratory, NY 11724.

FEBS Letters
|June 18, 1990
PubMed

Insights

Maturation promoting factor (MPF) activation in Xenopus oocytes involves cdc2 dephosphorylation. A p13-sensitive state precedes full cdc2 activation, which is inhibited by p13suc1 binding after tyrosine dephosphorylation.

Area of Science:

  • Cell Biology
  • Molecular Biology
  • Developmental Biology

Background:

  • Maturation promoting factor (MPF) activation during the prophase-metaphase transition in Xenopus oocytes is linked to the tyrosine dephosphorylation of the cdc2 protein.
  • Kinase activation in vivo and in cell-free extracts can be inhibited by excess p13suc1, a protein kinase subunit.

Purpose of the Study:

  • To investigate the role of p13suc1 in the activation of cdc2.
  • To define the p13-sensitive state in cdc2 activation following tyrosine dephosphorylation.

Main Methods:

  • In vitro activation of affinity-purified cdc2 from Xenopus prophase oocytes using potato acid phosphatase.
  • Assessing the effect of excess p13suc1 on cdc2 activation and tyrosine dephosphorylation in vitro.

Main Results:

  • Xenopus prophase cdc2 can be activated in vitro by potato acid phosphatase.
  • Excess p13suc1 does not inhibit tyrosine dephosphorylation of prophase cdc2 but prevents its activation by binding.
  • Fully activated metaphase Xenopus egg cdc2 is insensitive to excess p13suc1.

Conclusions:

  • A p13-sensitive state exists in cdc2 activation that occurs after tyrosine dephosphorylation.
  • p13suc1 binding to cdc2 defines a specific regulatory step in the activation process.

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