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Coronary artery-left ventricular microfistulae associated with apical hypertrophic cardiomyopathy
Ozgül Uçar1, Hülya Ciçekçioglu, Mustafa Cetin
1Department of Cardiology, Ankara Numune Education and Research Hospital, Talatpaşa Bulvari, Sihhiye, Ankara, Turkey.
Insights
A rare combination of coronary artery microfistulae and apical hypertrophic cardiomyopathy presented with angina pectoris. Beta-blocker therapy effectively alleviated symptoms in this unique case.
Area of Science:
- Cardiology
- Cardiovascular Medicine
- Medical Imaging
Background:
- Coronary artery to left ventricular microfistulae are rare vascular anomalies.
- Apical hypertrophic cardiomyopathy is a distinct form of hypertrophic cardiomyopathy.
Observation:
- A 58-year-old man presented with angina pectoris and ECG changes (ST depression, inverted T waves).
- Coronary angiography identified widespread coronary artery to left ventricular microfistulae.
- Left ventriculography and echocardiography confirmed apical hypertrophic cardiomyopathy.
Findings:
- The patient exhibited a rare co-occurrence of coronary artery microfistulae and apical hypertrophic cardiomyopathy.
- Angina pectoris symptoms were successfully managed with beta-blocker medication.
Implications:
- This case highlights the importance of considering rare conditions in complex cardiovascular presentations.
- Further research into the pathophysiology and clinical management of this combined condition is warranted.
- Understanding this rare combination may improve patient outcomes and therapeutic strategies.
Abstract:
A 58 year-old Caucasian man was admitted to the coronary care unit with angina pectoris. There were deep inverted T waves and ST segment depression at anterior precordial derivations. Coronary angiography revealed widespread coronary artery to left ventricular microfistulae arising from distal portions of both left and right coronary systems. Left ventriculography and transthoracic echocardiography revealed typical features of apical hypertrophic cardiomyopathy. Angina pectoris was alleviated by beta-blocker therapy. Both multiple coronary artery to left ventricular microfistulae and apical hypertrophic cardiomyopathy are rare conditions and little is known about pathophysiological and clinical aspects of this combination. Accumulating evidence will provide us this information so that the management of the patients will be enhanced.
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