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Degradation of human myelin in vitro by leucocytes from patients with multiple sclerosis

S J Owen1, C M Watson, A N Davison

  • 1Department of Neurochemistry, Institute of Neurology, London, England, U.K.

Insights

Peripheral blood cells from multiple sclerosis (MS) patients show significant myelin degradation activity, suggesting an autoimmune basis for MS. This cellular activity may indirectly reflect central nervous system inflammation.

Area of Science:

  • Neuroimmunology
  • Autoimmunity

Background:

  • Multiple sclerosis (MS) is a chronic inflammatory disease of the central nervous system.
  • The autoimmune basis of MS is under investigation, with a focus on myelin breakdown.

Purpose of the Study:

  • To quantitatively investigate human myelin breakdown in vitro to understand the autoimmune basis of MS.
  • To assess myelin degradative activity in peripheral blood mononuclear cells (PBMCs) from MS patients and controls.

Main Methods:

  • A quantitative method was employed to study human myelin breakdown in vitro.
  • PBMCs were isolated from patients with MS, rheumatoid arthritis (RA), other neurological diseases, and healthy controls.
  • Myelin degradative activity of PBMCs was measured.

Main Results:

  • Serum from MS patients and controls generally lacked myelin degradative activity.
  • Significant myelin degradative activity was observed in PBMCs from 43% of MS patients and 61.5% of RA patients.
  • Myelin degradation by cells was found in 13% of patients with other neurological diseases and in no healthy controls.

Conclusions:

  • Peripheral cellular immune activity demonstrates myelin degradative capacity, suggesting an autoimmune component in MS and RA.
  • This activity may originate from cells activated at primary inflammatory sites (CNS in MS, joints in RA).
  • Blood activity may indirectly reflect central inflammation, explaining the lack of correlation with clinical status in MS patients.

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