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PT pretreatment inhibits 48/80-induced activation of Ca(+)-permeable channels in rat peritoneal mast cells
1Department of Physiology, Osaka City University Medical School, Japan.
Abstract:
We recently reported that the secretagogue, compound 48/80, activated Ca2(+)-permeable channels of mast cells possibly via a second messenger [Kuno, Okada, and Shibata. Am. J. Physiol. 256 (Cell Physiol. 25): C560-C568, 1989]. The effects of pretreatment with pertussis toxin (PT) on compound 48/80 (48/80)-induced activation of the Ca2(+)-permeable channel have now been investigated by measuring Ca2+ signals of cell suspensions using the Ca2+ indicator fura-2 and by recording Ba2+ currents through the channel using the patch-clamp technique. In the presence of extracellular Ca2+, the fluorescence change was biphasic, with an immediate rise and a delayed peak at room temperature. The delayed peak, mainly due to Ca2+ entry through plasma membranes, was greatly reduced by pretreatment with PT. The quenching of the fluorescence by 48/80-induced Mn2+ influx was also decreased by PT, whereas the Ca2+ transients due to Ca2+ release from the intracellular stores apparently did not change. In patch-clamp recordings from cell-attached patches with pipettes containing isotonic Ba2+, the 48/80-induced Ba2+ currents were either suppressed or delayed in the PT-treated cells, under conditions where degranulation was absent. These results suggest that PT-sensitive GTP-binding protein is involved in activating the Ca2(+)-permeable channel in mast cells during stimulus-secretion coupling.
Insights
Pertussis toxin (PT) inhibits compound 48/80-induced calcium channel activation in mast cells. This suggests a role for PT-sensitive GTP-binding proteins in mast cell stimulus-secretion coupling.
Area of Science:
- Immunology
- Cell Biology
- Physiology
Background:
- Compound 48/80 activates calcium channels in mast cells, potentially through a second messenger.
- Mast cells play a crucial role in immune responses and degranulation.
Purpose of the Study:
- To investigate the effect of pertussis toxin (PT) pretreatment on compound 48/80-induced calcium channel activation in mast cells.
- To elucidate the involvement of signaling pathways in mast cell activation.
Main Methods:
- Measurement of intracellular calcium signals using fura-2 in cell suspensions.
- Recording of barium currents through calcium channels using the patch-clamp technique.
- Assessment of manganese influx as an indicator of calcium channel activity.
Main Results:
- PT pretreatment significantly reduced the delayed peak of intracellular calcium increase, primarily due to inhibited calcium influx.
- PT decreased compound 48/80-induced manganese influx, indicating reduced channel activity.
- Patch-clamp recordings showed suppressed or delayed barium currents in PT-treated cells.
- Calcium transients from intracellular stores were not significantly affected by PT.
Conclusions:
- A pertussis toxin-sensitive GTP-binding protein is implicated in the activation of calcium-permeable channels in mast cells.
- This finding highlights the role of G-protein signaling in mast cell stimulus-secretion coupling.