[Severe sepsis, septic shock and secondary multiple organ dysfunction in infection by Kluyvera ascorbata]

Raúl Carrillo Esper1, Carlos Peña Pérez, Jimena Muciño Bermejo

  • 1Servicio de Terapia Intensiva, Fundación Clínica Médica Sur, México. Revistacma95@yahoo.com.mx

Gaceta Medica De Mexico
|September 7, 2011
PubMed

Insights

Kluyvera ascorbata is an emerging pathogen causing severe sepsis. This case highlights the potential danger of this bacterium, even in young, otherwise healthy individuals.

Area of Science:

  • Microbiology
  • Infectious Diseases
  • Bacteriology

Background:

  • Kluyvera represents an emerging genus of Enterobacteriaceae, increasingly recognized as a potential pathogen.
  • Kluyvera species are found in various clinical specimens, including sputum, urine, stool, and blood.
  • These bacteria pose a risk due to their ability to cause diverse infections and transfer extended-spectrum beta-lactamase genes.

Observation:

  • A case report details a teenage male patient presenting with severe sepsis and septic shock.
  • The patient's condition was attributed to an infection caused by Kluyvera ascorbata.

Findings:

  • Kluyvera ascorbata was identified as the causative agent of severe sepsis and septic shock.
  • The case underscores the pathogenic potential of Kluyvera species.

Implications:

  • This case highlights the importance of recognizing Kluyvera species as significant pathogens, particularly in severe infections.
  • Understanding the clinical manifestations and transmission of Kluyvera is crucial for effective patient management.
  • Further research into the virulence factors and antimicrobial resistance of Kluyvera is warranted.

Related Concept Videos

Pneumonia III: Complications and Assessment01:30

Pneumonia III: Complications and Assessment

Pneumonia poses the potential for numerous complications that warrant consideration. These complications include the following:
Acute Pyelonephritis II: Diagnostic Studies and Management01:28

Acute Pyelonephritis II: Diagnostic Studies and Management

Introduction:For diagnosing acute pyelonephritis, a comprehensive patient history is collected to identify symptoms such as dysuria, frequent or urgent urination, flank pain, or costovertebral angle (CVA) tenderness that may suggest a kidney infection.Physical ExaminationDuring the physical examination, CVA tenderness is assessed. This involves gentle percussion over the costovertebral angle, where tenderness often indicates a kidney infection.Diagnostic TestsUrinalysis: Used to identify white...
Healthcare Associated Infections I: Iatrogenic, Exogenic and Endogenic01:26

Healthcare Associated Infections I: Iatrogenic, Exogenic and Endogenic

Healthcare-associated infections (HAIs) occur in a healthcare facility while a person receives care for another ailment. This category also includes work-related infections among healthcare staff.
HAIs significantly increase the cost of health care. Extended stays in healthcare institutions, increased disability, increased costs of medications, including specialized antibiotics, and prolonged recovery times add to the patient's expenses and the healthcare institution and funding bodies. Common...
Acute Kidney Injury III: Clinical Manifestations01:29

Acute Kidney Injury III: Clinical Manifestations

Acute Kidney Injury (AKI) progresses through distinct clinical phases: the oliguric, diuretic, and recovery phases, each marked by unique manifestations and challenges.Oliguric Phase:The oliguric phase is the initial stage of AKI, typically lasting 10 to 14 days. This phase is marked by a significant reduction in urine output, usually less than 400 mL per day, indicating decreased kidney function. Fluid retention is a prominent feature, leading to symptoms such as edema, hypertension, and...
Acute Inflammation III: Local and Systemic Effects01:25

Acute Inflammation III: Local and Systemic Effects

Acute inflammation produces a coordinated set of local and systemic changes that limit injury, eliminate pathogens, and initiate repair. These responses arise within minutes of infection, trauma, or chemical insult and are driven by vascular alterations and leukocyte-derived mediators. When the stimulus resolves, the reaction typically abates within days.Local EffectsAt the site of injury, arteriolar vasodilation increases blood flow, resulting in redness and warmth. Simultaneously, increased...
Endocarditis II: Clinical Features of Infective Endocarditis01:25

Endocarditis II: Clinical Features of Infective Endocarditis

Endocarditis can present various clinical features depending on the causative organism and the patient's underlying health conditions. Initially, the clinical features of infective endocarditis develop gradually, presenting with nonspecific symptoms that can be easily mistaken for other illnesses.General SymptomsEarly symptoms of infective endocarditis are fever, chills, weakness, malaise, fatigue, and weight loss. These symptoms reflect the systemic nature of the infection and the body's...