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Related Experiment Videos

Superficial thrombophlebitis. I. Primary hypercoagulable states.

C P Samlaska1, W D James

  • 1Dermatology Service, Walter Reed Army Medical Center, Washington, D.C.

Journal of the American Academy of Dermatology
|June 1, 1990
PubMed
Summary
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Superficial thrombophlebitis can signal primary hypercoagulable states, often inherited defects in coagulation or fibrinolysis. This review covers their physiology, clinical signs, and treatments.

Area of Science:

  • Hematology
  • Vascular Medicine
  • Genetics

Background:

  • Superficial thrombophlebitis can be a key clinical indicator of underlying hypercoagulable states.
  • Primary hypercoagulable states involve specific, measurable defects in the coagulation and fibrinolytic systems.
  • These conditions increase the risk of thrombosis.

Purpose of the Study:

  • To review disorders where superficial thrombophlebitis is a significant clinical sign.
  • To detail the physiology, pathophysiology, clinical characteristics, and treatment of primary hypercoagulable states.

Main Methods:

  • Literature review focusing on primary hypercoagulable states.
  • Analysis of inherited and acquired conditions predisposing to thrombosis.
  • Examination of clinical presentations and management strategies.

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Main Results:

  • Identified inherited deficiencies (antithrombin III, heparin cofactor 2, protein C, protein S) as common causes.
  • Included abnormal fibrinolytic activity, dysfibrinogenemia, and Hageman trait.
  • Recognized lupus anticoagulant/anticardiolipin antibody syndrome as a primary hypercoagulable state.

Conclusions:

  • Superficial thrombophlebitis warrants investigation for underlying hypercoagulable disorders.
  • Understanding these defects is crucial for effective thrombosis prevention and treatment.
  • Comprehensive review aids in diagnosing and managing patients with thrombotic risks.