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Mechanisms by which EGF receptor and TGF alpha contribute to malignant transformation

E Di Marco1, J H Pierce, S A Aaronson

  • 1Laboratory of Cellular and Molecular Biology, National Cancer Institute, National Institutes of Health, Bethesda, Md.

Natural Immunity and Cell Growth Regulation
|January 1, 1990
PubMed

Insights

Alterations in the epidermal growth factor pathway are common in cancers. This review explores how growth factors and their receptors contribute to tumor development using in vitro models.

Area of Science:

  • Oncology
  • Molecular Biology
  • Cell Signaling

Background:

  • Aberrant signaling through the epidermal growth factor (EGF) pathway is a hallmark of many cancers.
  • Overexpression of the EGF receptor and production of transforming growth factor alpha (TGF-α) are frequently observed in tumor cells.
  • These molecular alterations suggest a role in malignant transformation.

Purpose of the Study:

  • To review the establishment of in vitro model systems for studying the transforming potential of EGF pathway molecules.
  • To summarize current knowledge on the mechanisms driving transformation mediated by growth factor and growth factor receptor genes.

Main Methods:

  • Utilized in vitro model systems to investigate the functional consequences of altered EGF pathway signaling.
  • Reviewed existing literature on the role of growth factors and receptors in cancer development.

Main Results:

  • Demonstrated the utility of in vitro models in dissecting the transforming potential of specific genes.
  • Highlighted the distinct expression patterns of TGF-α in tumor versus normal cells.
  • Summarized key mechanisms by which growth factor and receptor genes contribute to cellular transformation.

Conclusions:

  • In vitro models are crucial for understanding the oncogenic roles of growth factors and receptors.
  • The EGF/TGF-α pathway represents a significant target for cancer research and therapeutic intervention.

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