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Presenilin-2 modulation of ER-mitochondria interactions: FAD mutations, mechanisms and pathological consequences

Enrico Zampese1, Cristina Fasolato, Tullio Pozzan

  • 1Department of Biomedical Sciences; University of Padova and CNR Institute of Neuroscience; Padova, Italy.

Communicative & Integrative Biology
|October 8, 2011
PubMed

Keywords:
Alzheimer's DiseaseCa2+endoplasmic reticulummitochondriaorganelle juxtapositionpresenilin

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