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Updated: May 28, 2026

Point-of-Care Ultrasound for Peripheral Veno-Arterial Extracorporeal Membrane Oxygenation Without Left Ventricular Venting
Published on: January 17, 2025
Transseptal catheter decompression of the left ventricle during extracorporeal membrane oxygenation
Michael F Swartz1, Frank Smith, Craig J Byrum
1Department of Surgery, Strong Memorial Hospital, University of Rochester, Rochester, NY 14582, USA.
Insights
Veno-arterial extracorporeal membrane oxygenation (VA-ECMO) can cause left ventricular distension. A transseptal cannula effectively decompressed the left ventricle, improving patient outcomes and enabling VA-ECMO weaning.
Area of Science:
- Cardiology
- Critical Care Medicine
- Pediatric Cardiology
Background:
- Veno-arterial extracorporeal membrane oxygenation (VA-ECMO) is a life-support measure for severe cardiogenic shock.
- Left ventricular distension and pulmonary edema are known complications of VA-ECMO.
- Effective left ventricular unloading is crucial for successful VA-ECMO support.
Observation:
- A 13-year-old girl with cardiogenic shock required VA-ECMO.
- After 6 days, severe left ventricular distension impeded VA-ECMO function.
- Bedside transesophageal echocardiography guided the intervention.
Findings:
- A percutaneous atrial transseptal cannula was inserted to decompress the left ventricle.
- The transseptal cannula was connected to the venous circuit, facilitating left ventricular unloading.
- The patient demonstrated clinical improvement following the intervention.
Implications:
- Percutaneous transseptal catheterization is a viable strategy for managing VA-ECMO-induced left ventricular distension.
- This technique can improve VA-ECMO circuit flows and patient recovery.
- It offers a minimally invasive approach to address a critical complication in pediatric cardiac support.
Abstract:
Veno-arterial extracorporeal membrane oxygenation (VA-ECMO) can result in left-ventricular distension and the development of pulmonary edema. We present the case of a 13-year-old girl who presented with cardiogenic shock. VA-ECMO was initiated, but after 6 days, severe left-ventricular distension resulted in decreased VA-ECMO flows. With guidance by bedside transesophageal echocardiography, a percutaneous atrial transseptal cannula was placed and connected to the venous circuit, thus decompressing the left ventricle. The patient improved, was weaned from VA-ECMO 5 days later, and was discharged from the hospital. Bedside transseptal catheter insertion is an effective method of left-ventricular decompression.
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