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Molecular characterization of trimethoprim resistance in Shigella sonnei in Sicily

A Agodi1, C Jones, E J Threlfall

  • 1Institute of Hygiene and Preventive Medicine, University of Catania, Italy.

Insights

Shigella sonnei strains in Sicily showed high trimethoprim resistance linked to specific plasmids. This resistance was associated with the dihydrofolate reductase (DHFR) type V gene, newly reported outside Sri Lanka.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Antimicrobial Resistance

Background:

  • Shigella sonnei infections can lead to antibiotic resistance.
  • Trimethoprim resistance in bacteria is a growing public health concern.
  • Understanding the genetic basis of antibiotic resistance is crucial for effective treatment.

Purpose of the Study:

  • To investigate the prevalence and genetic basis of trimethoprim resistance in Shigella sonnei strains from Catania, Sicily.
  • To characterize the plasmids associated with trimethoprim resistance.
  • To identify the specific dihydrofolate reductase (DHFR) gene type responsible for resistance.

Main Methods:

  • Plasmid analysis using electropherotyping.
  • Restriction endonuclease fingerprinting of plasmid DNA.
  • DNA hybridization analysis using DHFR gene probes.

Main Results:

  • All Shigella sonnei strains isolated between 1985-1987 exhibited high trimethoprim resistance.
  • Most resistant strains harbored plasmids of 70 MDa and/or 130 MDa.
  • DNA hybridization confirmed the presence of the DHFR type V gene in strains with transferable trimethoprim resistance, with no detectable DHFR types I, II, or IV.

Conclusions:

  • The DHFR type V gene is associated with trimethoprim resistance in Shigella sonnei in Sicily.
  • This finding represents the first report of the DHFR type V gene outside of Sri Lanka.
  • The study highlights the importance of plasmid-mediated resistance mechanisms in the spread of antibiotic resistance.

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