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Diet, central nervous system, and aging.

L D Lytle, A Altar

    Federation Proceedings
    |May 1, 1979
    PubMed
    Summary
    This summary is machine-generated.

    Aging impacts the central nervous system (CNS) with brain atrophy and impaired neuronal communication. Nutritional factors may influence these geriatric changes in the aging brain.

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    Area of Science:

    • Neuroscience
    • Gerontology
    • Biochemistry

    Background:

    • Aging is associated with significant morphological, structural, and chemical alterations in the central nervous system (CNS).
    • Brain atrophy, characterized by reduced size and volume, occurs during senescence.
    • Changes in nerve and glial cell characteristics and impaired neurotransmission are observed in the aging brain.

    Purpose of the Study:

    • To explore the multifaceted changes in the aging central nervous system.
    • To investigate the potential role of nutritional factors in CNS aging.
    • To understand how nutrition influences brain neurotransmission and structural changes during senescence.

    Main Methods:

    • Review of existing literature on CNS aging.
    • Analysis of morphological, structural, and chemical changes in aging brains.

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  • Examination of evidence on neurotransmitter function in elderly subjects.
  • Assessment of nutritional influences on brain aging.

    • Main Results:

    • Aging leads to brain atrophy and alterations in neuronal and glial cell populations.
    • Evidence suggests impaired communication between CNS cells in the elderly.
    • Nutritional factors show potential to modulate brain neurotransmission.
    • Nutrition may accelerate or retard age-related structural changes in the CNS.

    Conclusions:

    • The aging central nervous system undergoes substantial changes affecting structure and function.
    • Nutritional interventions may represent a key strategy to mitigate negative impacts of aging on the brain.
    • Further research is warranted to elucidate the precise mechanisms of nutritional influence on CNS aging.