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Bronchorrhea following Stroke.

Chung-Yang Sung1, Tsong-Hai Lee, Nai-Shin Chu

  • 1Department of Neurology, Chang Gung Memorial Hospital, Taoyuan, Taiwan, ROC. sung0597@cgmh.org.tw

European Neurology
|December 20, 2011
PubMed
Summary
This summary is machine-generated.

Stroke can cause excessive bronchial secretion (bronchorrhea), particularly with dorsal medulla lesions. This condition is treatable with anticholinergic medication, improving patient outcomes.

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Area of Science:

  • Neurology
  • Pulmonology

Background:

  • Stroke-induced bronchorrhea is an under-recognized complication.
  • Excessive bronchial secretion following stroke requires further investigation for its pathophysiology and management.

Purpose of the Study:

  • To investigate the clinical characteristics, pathophysiology, and treatment of stroke-induced bronchorrhea.
  • To highlight the association between dorsal medulla lesions and bronchorrhea.

Main Methods:

  • Prospective data collection from 19 stroke patients with bronchorrhea.
  • Analysis of stroke type, lesion location, secretion onset, severity, duration, and treatment response.

Main Results:

  • 84% ischemic stroke, 16% hemorrhagic; all had brainstem involvement, 58% with dorsal lateral medulla lesions.
  • Copious secretion in 79% within 3 days, lasting 4 days to over 2 months.
  • Anticholinergic treatment in 53% showed fair to good response.

Conclusions:

  • Dorsal medulla involvement in stroke may cause bronchorrhea, likely mediated by parasympathetic pathways.
  • Early diagnosis and anticholinergic treatment can alleviate suffering and prevent complications.
  • Effective management reduces antibiotic use, aspiration pneumonia risk, and hospitalization duration.