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Isolation of a Friend recombinant polytropic virus with a T-cell-restricted leukemogenicity

V Della-Valle1, F Wendling, M Sitbon

  • 1U-301 INSERM, Institute de Génétique Moléculaire, Paris, France.

Virology
|October 1, 1990
PubMed

Insights

Researchers isolated novel polytropic viruses from F-MuLV infected mice. One isolate, EA1-17, uniquely caused T-cell leukemia in recipient mice, offering new insights into viral oncogenesis.

Area of Science:

  • Virology
  • Oncology
  • Immunology

Background:

  • Friend Murine Leukemia Virus (F-MuLV) is known to cause hematopoietic malignancies.
  • Recombinant polytropic viruses (MCF or dualtropic viruses) can arise during F-MuLV infection.
  • Understanding viral tropism is crucial for identifying oncogenic mechanisms.

Purpose of the Study:

  • To isolate and characterize novel recombinant polytropic viruses from F-MuLV infected mice.
  • To investigate the oncogenic potential and tropism of these isolated viruses.

Main Methods:

  • Isolation of hematopoietic malignant cells from F-MuLV inoculated mice.
  • In vivo cell transplantation and serial passaging in recipient mice.
  • Isolation of recombinant polytropic viruses using limiting dilution.
  • Inoculation of isolated viruses into newborn mice to assess pathogenicity and tropism.

Main Results:

  • Several recombinant polytropic viruses were successfully isolated from tumors.
  • Two isolates, RA1-17 and EA1-17, were obtained from the same animal.
  • RA1-17 induced erythroblastosis, consistent with known Friend recombinant polytropic viruses.
  • EA1-17 demonstrated a novel T-cell leukemia tropism with a short latency period.

Conclusions:

  • The isolation of EA1-17 represents the first polytropic recombinant virus with T-cell tropism linked to F-MuLV infection.
  • This finding expands the known spectrum of F-MuLV-induced leukemias.
  • These novel viral isolates provide valuable tools for studying viral oncogenesis and host-pathogen interactions.

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