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Related Experiment Videos

G6PD deficiency in neonates: a prospective study.

M Verma1, D Singla, S B Crowell

  • 1Department of Pediatrics, Christian Medical College, Ludhiana, Punjab.

Indian Journal of Pediatrics
|May 1, 1990
PubMed
Summary
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Glucose-6-phosphate dehydrogenase (G6PD) deficiency affects 3.9% of newborns, with higher rates in males. Screening neonates for G6PD deficiency is crucial for diagnosing jaundice and preventing hemolytic anemia.

Area of Science:

  • Biochemistry
  • Pediatrics
  • Genetics

Background:

  • Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a common inherited condition.
  • Neonatal jaundice is a frequent clinical concern in newborns.

Purpose of the Study:

  • To determine the frequency of G6PD deficiency in a cohort of newborns.
  • To investigate the association between G6PD deficiency and neonatal jaundice.
  • To assess the utility of G6PD screening in neonates presenting with jaundice.

Main Methods:

  • Screening of 1000 consecutively born babies for G6PD deficiency.
  • Observation of neonates for seven days for jaundice development.
  • Parental screening for G6PD deficiency in affected infants.

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Main Results:

  • The overall frequency of G6PD deficiency was 3.9% (5% in males, 2.8% in females).
  • Religion did not significantly impact the deficiency frequency.
  • Hyperbilirubinemia developed in 48.7% of neonates with G6PD deficiency.
  • Maternal G6PD deficiency was common among infants with the condition.

Conclusions:

  • Neonatal G6PD deficiency is prevalent and linked to hyperbilirubinemia.
  • Screening for G6PD deficiency in jaundiced neonates is recommended.
  • Early detection of G6PD deficiency aids in preventing hemolytic episodes.