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Septicaemia and the prevention of multiorgan failure--the intensive care perspective

K Crowley1, D Phelan

  • 1Department of Anaesthesia and Intensive Care, Mater Hospital, Dublin.

Irish Medical Journal
|September 1, 1990
PubMed

Insights

Septicaemia, a severe infection, often lacks typical symptoms and has high mortality. Microvascular failure leading to tissue hypoxia is a key factor in organ failure and death, necessitating supportive circulatory management.

Area of Science:

  • Critical Care Medicine
  • Infectious Diseases
  • Pathophysiology

Background:

  • Septicaemia presents atypically, with tachypnoea, hypotension, and confusion as common signs.
  • High mortality rates (40-80%) and significant ICU deaths (70%) highlight the severity of septicaemia.
  • Despite sensitive antimicrobial drugs, patient mortality remains high due to mediator-induced organ system effects.

Purpose of the Study:

  • To explore the unifying hypothesis of microvascular failure in septicaemia-induced multiple organ failure.
  • To discuss the correlation between organ system failures and mortality in septicaemia.
  • To outline current supportive management strategies focused on preventing organ failure.

Main Methods:

  • Focus on supportive management, emphasizing circulatory manipulation.
  • Utilizing intravascular volume expansion, vasoactive drugs, mechanical ventilation, and invasive monitoring.
  • Employing a 'best guess' antimicrobial approach with multiple agents pending organism isolation.

Main Results:

  • Microvascular failure causing tissue hypoxia is proposed as the unifying mechanism for multiple organ failure.
  • Mortality is directly correlated with the number of organ systems failing.
  • Aggressive surgical drainage or excision is crucial for abdominal sepsis.

Conclusions:

  • Supportive management, particularly circulatory support, is central to preventing organ failure in septicaemia.
  • Antimicrobial therapy requires a pragmatic 'best guess' strategy initially.
  • Experimental adjunctive therapies targeting sepsis mediators show potential but require further investigation.

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