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Alterations in the pathogenicity of one Paracoccidioides brasiliensis isolate do not correlative with its in vitro

S S Kashino1, L M Singer-Vermes, V L Calich

  • 1Depto. Imunologia, Universidade de São Paulo, Brasil.

Mycopathologia
|September 1, 1990
PubMed

Insights

Continuous in vitro cultivation of Paracoccidioides brasiliensis (Pb 18) reduces its pathogenicity, but reisolation from mice restores virulence. In vitro growth patterns did not correlate with pathogenicity changes.

Area of Science:

  • Medical Mycology
  • Infectious Diseases
  • Microbial Pathogenesis

Background:

  • Long-term in vitro subcultivation of microorganisms can lead to attenuation of pathogenicity.
  • Reisolation from a host organism can potentially restore virulence in attenuated microbial strains.

Purpose of the Study:

  • To compare the pathogenicity and in vitro growth patterns of Paracoccidioides brasiliensis (Pb 18) isolate.
  • To investigate the effects of continuous in vitro subcultivation and subsequent reisolation from mice on Pb 18 virulence.

Main Methods:

  • Comparison of original pathogenic (OP), in vitro attenuated (AT), and reisolated (RS, RR) Pb 18 yeast phase isolates.
  • Pathogenicity assessment via mouse mortality and median survival times after intraperitoneal infection.
  • In vitro growth curve analysis to determine total and viable yeast counts.

Main Results:

  • Continuous in vitro subcultivation of Pb 18 led to decreased pathogenicity, indicated by increased median survival times in infected mice.
  • Reisolation of Pb 18 from both susceptible (RS) and resistant (RR) mice restored pathogenicity, with survival times similar to the original strain.
  • The reisolated (RR) variant exhibited significantly higher in vitro yeast counts compared to other variants, but this did not correlate with pathogenicity.

Conclusions:

  • Paracoccidioides brasiliensis isolate Pb 18 loses pathogenicity with prolonged in vitro subcultivation, which is reversible upon reisolation from infected mice.
  • In vitro growth characteristics of Pb 18 are influenced by the host environment but do not directly correlate with observed changes in pathogenicity.

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