Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Cranial and Spinal Meninges01:19

Cranial and Spinal Meninges

The cranial and spinal meninges are complex protective structures surrounding the central nervous system (CNS), consisting of the brain and spinal cord. These meninges consist of the dura mater, the arachnoid mater, and the pia mater. They protect the CNS, provide structural support, and aid in circulating cerebrospinal fluid (CSF).
Cranial Meninges
These meningeal layers cover the cranium. The dura mater is the outermost layer of cranial meninges. It is a thick and durable membrane of dense...
Increased Intracranial Pressure l: Introduction01:14

Increased Intracranial Pressure l: Introduction

Intracranial hypertension is a sustained elevation of intracranial pressure (ICP) above 22 mm Hg. In supine adults, normal ICP is ~7–15 mm Hg.The rigid, nonexpandable cranium contains three components—brain tissue, blood, and cerebrospinal fluid (CSF)—that total ~1,700 mL in a typical adult: 1,400 mL brain (~80%), 150 mL blood (~10%), and 150 mL CSF (~10%). According to the Monro–Kellie doctrine, total intracranial volume is effectively fixed. When one component expands, CSF and venous blood...
Brain Abscess l: Introduction01:26

Brain Abscess l: Introduction

A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial infections,...
Anatomy of the Brain: Ventricles01:18

Anatomy of the Brain: Ventricles

There are hollow fluid-filled cavities known as ventricles deep inside the human brain. There are two lateral ventricles, one in each cerebral hemisphere, and each has three different projections — the anterior, inferior, and posterior horns visible from the lateral side. A thin membrane called the septum pellucidum separates the two lateral ventricles. The slender third ventricle in the diencephalon is connected to each lateral ventricle via a channel called the interventricular foramen. The...
Cerebral Edema ll: Pathophysiology01:22

Cerebral Edema ll: Pathophysiology

Vasogenic edema is a major form of cerebral edema characterized by abnormal accumulation of fluid in the brain’s extracellular space due to disruption of the blood–brain barrier (BBB). The BBB is a specialized structure composed of endothelial cells connected by tight junctions, supported by astrocytic endfeet and a basement membrane. Under normal conditions, it tightly regulates the movement of ions, proteins, and solutes between the bloodstream and brain parenchyma. When this barrier loses...
Increased Intracranial Pressure ll: Pathophysiology01:29

Increased Intracranial Pressure ll: Pathophysiology

Increased intracranial pressure (ICP) refers to a potentially life-threatening rise in pressure inside the skull. This usually happens when there is a major change in the volume of brain tissue, blood, or cerebrospinal fluid (CSF) — the three components inside the skull. According to the Monro-Kellie doctrine, if the volume of one component increases, the volumes of the other components must decrease to maintain normal pressure. If this does not happen, ICP rises.The process often begins with...

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Novel CAR T cell blend targeting PDPN and GD2 to overcome glioblastoma heterogeneity.

Journal for immunotherapy of cancer·2026
Same author

Guidelines for Neuroprognostication in Critically ill Adults with Acute Ischemic Stroke.

Neurocritical care·2026
Same author

Guidelines for Neuroprognostication in Critically Ill Adults with Status Epilepticus.

Neurocritical care·2026
Same author

Low tumour burden is associated with observation after surgery in patients with grade 2 astrocytoma and oligodendroglioma: results from the prospective multicentre LoG-Glio registry.

Journal of neuro-oncology·2025
Same author

Niels Sörensen (September 2, 1941-March 6, 2025)-founder of pediatric neurosurgery in Germany.

Child's nervous system : ChNS : official journal of the International Society for Pediatric Neurosurgery·2025
Same author

Pathogenic Variants and Allele Loss of the NF2 and LZTR1 Gene in Sporadic Vestibular Schwannoma.

In vivo (Athens, Greece)·2025
Same journal

Peptidomics in the Spotlight: Advanced Sample Treatment Techniques and Analytical Insights.

Advances in experimental medicine and biology·2026
Same journal

Methods for the Investigation of Protein-Ligands Interactions.

Advances in experimental medicine and biology·2026
Same journal

Sample Preparation Strategies for Microbial Cell Surface Proteomics: Integrating Shaving and Shotgun Approaches.

Advances in experimental medicine and biology·2026
Same journal

Proteomic Sample Preparation for the Petroleum Industry: A Biocorrosion Case Study.

Advances in experimental medicine and biology·2026
Same journal

Proteomic and Functional Comparison of Extracellular Vesicles from Wild-Type and Lyn-Deficient Stromal Cells.

Advances in experimental medicine and biology·2026
Same journal

Proteomic Analysis of Histone Sequence Variants and Post-translationally Modified Forms.

Advances in experimental medicine and biology·2026
See all related articles

Related Experiment Video

Updated: May 24, 2026

Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage
10:34

Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage

Published on: August 30, 2020

Arachnoid cysts.

Thomas Westermaier1, Tilmann Schweitzer, Ralf-Ingo Ernestus

  • 1Department of Neurosurgery, University of Wuerzburg, Wuerzburg, Germany. westermaier.t@nch.uni-wuerzburg.de

Advances in Experimental Medicine and Biology
|March 14, 2012
PubMed
Summary
This summary is machine-generated.

Arachnoid cysts, fluid-filled sacs, are congenital defects, not neurodegenerative. While often asymptomatic, growing cysts may necessitate neurosurgical intervention for neurological symptoms.

More Related Videos

Endoscopic Approach for Colloid Cyst Resection
02:30

Endoscopic Approach for Colloid Cyst Resection

Published on: May 23, 2025

Related Experiment Videos

Last Updated: May 24, 2026

Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage
10:34

Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage

Published on: August 30, 2020

Endoscopic Approach for Colloid Cyst Resection
02:30

Endoscopic Approach for Colloid Cyst Resection

Published on: May 23, 2025

Area of Science:

  • Neurology
  • Developmental Biology
  • Neurosurgery

Background:

  • Arachnoid cysts are fluid-filled sacs arising from splitting or duplication of the arachnoid layer.
  • These cysts contain fluid similar but not identical to cerebrospinal fluid.
  • They are considered congenital defects of the arachnoid layer, not primary neurodegenerative disorders.

Purpose of the Study:

  • To explore the pathogenesis and clinical manifestations of arachnoid cysts.
  • To discuss the indications and options for neurosurgical treatment.
  • To address the controversial aspects of management strategies for arachnoid cysts.

Main Methods:

  • Review of literature on arachnoid cyst pathogenesis.
  • Analysis of clinical symptoms associated with arachnoid cysts.
  • Evaluation of neurosurgical treatment modalities and outcomes.

Main Results:

  • Arachnoid cysts can be congenital or develop de novo, varying in size.
  • Many arachnoid cysts are asymptomatic and discovered incidentally.
  • Symptomatic cysts can cause headaches, seizures, and other neurological deficits, requiring intervention.

Conclusions:

  • Management strategies for arachnoid cysts remain controversial.
  • Conservative management with imaging follow-up is suitable for asymptomatic or mildly symptomatic cases.
  • Neurosurgical treatment is indicated for growing or significantly symptomatic arachnoid cysts.