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Renal response to furosemide in very low birth weight infants during chronic administration

M H Mirochnick1, J J Miceli, P A Kramer

  • 1Department of Pediatrics, University of Connecticut, Farmington.

Developmental Pharmacology and Therapeutics
|January 1, 1990
PubMed

Insights

Premature infants with bronchopulmonary dysplasia show reduced kidney response to furosemide with chronic use and increasing age. This indicates a need to adjust diuretic therapy for these vulnerable infants.

Area of Science:

  • Neonatal Medicine
  • Pediatric Nephrology
  • Pharmacology

Background:

  • Bronchopulmonary dysplasia (BPD) is a common respiratory complication in premature infants.
  • Diuretics, such as furosemide, are frequently used in BPD management to reduce fluid overload.
  • Understanding the renal response to furosemide in this population is crucial for optimizing treatment.

Purpose of the Study:

  • To investigate the renal response to both initial and chronic doses of furosemide in premature infants with BPD.
  • To determine how renal sensitivity to furosemide changes with sustained use and increasing postconceptional age.

Main Methods:

  • Seven premature infants with BPD were studied.
  • Urine collections were performed over 12-hour periods after initial furosemide doses and after 1 and 3 weeks of chronic therapy.
  • Urinary output, furosemide excretion, sodium, and creatinine concentrations were measured.

Main Results:

  • A linear dose-response relationship was observed between urinary furosemide excretion and diuretic/natriuretic effects.
  • Higher furosemide excretion rates were needed for similar responses during chronic dosing compared to initial doses, indicating decreased renal responsiveness.
  • Increased postconceptional age at therapy initiation was associated with reduced initial furosemide responsiveness.

Conclusions:

  • Renal sensitivity to furosemide decreases with chronic administration in premature infants with BPD.
  • Increasing postconceptional age also correlates with diminished initial renal responsiveness to furosemide.
  • These findings suggest renal adaptation to chronic furosemide therapy, necessitating potential dose adjustments.

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