Resveratrol induces apoptosis associated with mitochondrial dysfunction in bladder carcinoma cells

Xi Lin1, Gang Wu, Wen-Qian Huo

  • 1Department of Urology, Daping Hospital, The Third Military Medical University, Chongqing, China.

Abstract

Insights

Resveratrol effectively reduces bladder cancer cell viability and triggers apoptosis by disrupting mitochondrial function. This natural compound activates the intrinsic mitochondrial pathway, showing promise for bladder cancer treatment.

Area of Science:

  • Oncology
  • Biochemistry
  • Cell Biology

Background:

  • Resveratrol exhibits chemopreventive properties against various human cancers.
  • Its known mechanisms involve targeting mitochondria and inducing apoptosis.

Purpose of the Study:

  • To investigate the antitumor effects of resveratrol on bladder cancer cells.
  • To elucidate the underlying molecular mechanisms, focusing on mitochondrial pathways.

Main Methods:

  • Cytotoxicity assessed using MTT assay in BTT739 and T24 bladder cell lines.
  • Apoptosis induction evaluated via transferase dUTP nick end labeling (TUNEL) staining.
  • Mitochondrial function analyzed through membrane potential, reactive oxygen species (ROS), and adenosine 5'-triphosphate (ATP) levels.
  • Intrinsic apoptosis pathway markers, including cytochrome c release and caspase-9/caspase-3 activity, were measured.

Main Results:

  • Resveratrol demonstrated a dose- and time-dependent decrease in cell viability and induced apoptosis.
  • Significant disruption of mitochondrial membrane potential was observed.
  • Increased ROS production and decreased ATP content were noted.
  • Resveratrol promoted cytochrome c release from mitochondria and activated caspase-9 and caspase-3.

Conclusions:

  • Resveratrol induces apoptosis in bladder cancer cells via the intrinsic mitochondrial pathway.
  • Mitochondrial dysfunction is a key mechanism in resveratrol's antitumor action.
  • Resveratrol holds significant therapeutic potential for bladder cancer treatment.

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