Cardiotoxic local anesthetics increasingly interact with biomimetic membranes under ischemia-like acidic conditions

Hironori Tsuchiya1, Maki Mizogami, Takahiro Ueno

  • 1Department of Dental Basic Education, School of Dentistry, Asahi University, Mizuho, Gifu, Japan. hiro@dent.asahi-u.ac.jp

Insights

Local anesthetics like bupivacaine and lidocaine become more cardiotoxic in acidic conditions. They interact more with cardiac mitochondrial membranes at low pH, potentially explaining toxicity during cardiac ischemia.

Area of Science:

  • Biochemistry
  • Pharmacology
  • Cardiovascular Research

Background:

  • Local anesthetics (LAs) can cause cardiotoxicity, particularly during cardiac ischemia.
  • Ischemia leads to acidosis, lowering tissue pH significantly.
  • Besides channel blockade, LA interaction with cell membranes is a proposed cardiotoxic mechanism.

Purpose of the Study:

  • To investigate the interaction of bupivacaine and lidocaine with cardiac mitochondrial membranes under acidic conditions mimicking ischemia.
  • To test the hypothesis that altered membrane interactions contribute to enhanced LA cardiotoxicity in acidosis.

Main Methods:

  • Utilized biomimetic membranes (unilamellar vesicles) composed of various phospholipids and cholesterol.
  • Adjusted buffer pH to simulate physiological (7.4) and ischemic (6.9, 6.4, 5.9) conditions.
  • Measured drug-membrane interactions using fluorescence polarization at cardiotoxically relevant LA concentrations.

Main Results:

  • Bupivacaine and lidocaine increased membrane fluidity in general phospholipid and nerve/cardiomyocyte-mimetic membranes, with reduced effect at lower pH.
  • In cardiomyocyte mitochondria-mimetic membranes with cardiolipin, LAs *increased* membrane interaction at acidic pH (5.9-6.4) compared to pH 7.4.
  • This effect was more pronounced in anionic phospholipid membranes, suggesting ion-pairing between positively charged LAs and negatively charged phospholipids.

Conclusions:

  • Local anesthetic interaction with cardiac mitochondrial membranes is pH-dependent.
  • Acidosis, characteristic of cardiac ischemia, enhances LA interaction with anionic phospholipids in mitochondrial membranes.
  • This pH-dependent membrane interaction may contribute significantly to the increased cardiotoxicity of local anesthetics observed in acidosis during cardiac ischemia.

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