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Updated: May 20, 2026

Studying Triple Negative Breast Cancer Using Orthotopic Breast Cancer Model
Published on: March 20, 2020
Triple-negative breast cancer: are we making headway at least?
Monica Arnedos1, Celine Bihan, Suzette Delaloge
1Breast Unit, Department of Medicine, Institut Gustave Roussy, Villejuif, France.
Abstract:
The so-called triple-negative breast cancer, as defined by tumors that lack estrogen receptor, progesterone receptor and human epidermal growth factor receptor 2 (HER2) overexpression, has generated growing interest in recent years despite representing less than 20% of all breast cancers. These tumors constitute an important clinical challenge, as they do not respond to endocrine treatment and other targeted therapies. As a group they harbor an aggressive clinical phenotype with early development of visceral metastases and a poor long-term prognosis. While chemotherapy remains effective in triple-negative disease, research continues to further identify potential new targets based on phenotypical and molecular characteristics of these tumors. In this respect, the presence of a higher expression of different biomarkers including epidermal growth factor receptor, vascular endothelial growth factor receptor, fibroblast growth factor receptor and Akt activation has led to a proliferation of clinical trials assessing the role of inhibitors to these pathways in triple-negative tumors. Moreover, the described overlap between triple-negative and basal-like tumors, and the similarities with tumors arising in the BRCA1 mutation carriers has offered potential therapeutic avenues for patients with these cancers including poly (ADP-ribose) polymerase inhibitors and a focus on a higher sensitivity to alkylating chemotherapy agents. Results from these trials have shown some benefit in small subgroups of patients, even in single-agent therapy, which reflects the heterogeneity of triple-negative breast cancer and highlights the need for a further subclassification of these types of tumors for better prognosis identification and treatment individualization.
Insights
Triple-negative breast cancer (TNBC) lacks common targets, presenting a clinical challenge. Research is identifying new therapeutic targets and subclassifications for personalized treatment of this aggressive cancer.
Area of Science:
- Oncology
- Molecular Biology
- Cancer Research
Background:
- Triple-negative breast cancer (TNBC) lacks estrogen receptor, progesterone receptor, and HER2 overexpression.
- TNBC represents less than 20% of breast cancers but has an aggressive phenotype and poor prognosis.
- Standard therapies are limited, necessitating research into novel treatment strategies.
Purpose of the Study:
- To review current research on therapeutic targets and treatment strategies for TNBC.
- To highlight the heterogeneity of TNBC and the need for further subclassification.
- To explore potential new treatment avenues based on molecular characteristics.
Main Methods:
- Review of clinical trials and research on TNBC biomarkers.
- Analysis of molecular similarities between TNBC, basal-like tumors, and BRCA1-mutated cancers.
- Evaluation of targeted therapies and chemotherapy agents in TNBC.
Main Results:
- Targeted therapies inhibiting pathways like EGFR, VEGFR, FGFR, and Akt show promise in subgroups.
- Poly (ADP-ribose) polymerase inhibitors and alkylating agents are potential therapeutic avenues.
- Clinical trial results indicate benefit in specific patient subgroups, underscoring TNBC heterogeneity.
Conclusions:
- TNBC is a heterogeneous disease requiring further subclassification for personalized treatment.
- Targeted therapies and novel agents offer potential benefits for specific TNBC patient subgroups.
- Continued research into molecular characteristics is crucial for improving TNBC prognosis and treatment individualization.
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