Mutation and genomic amplification of the PIK3CA proto-oncogene in pituitary adenomas

C B Murat1, P B S Braga, M A H Z Fortes

  • 1Laboratório de Endocrinologia Celular e Molecular (LIM-25), Universidade de São Paulo, São Paulo, SP, Brasil.

Insights

Genetic alterations in the PIK3CA gene, including mutations and amplification, are present in pituitary adenomas. This finding supports the PI3K/Akt pathway

Area of Science:

  • Endocrinology
  • Oncology
  • Molecular Biology

Background:

  • Pituitary adenoma tumorigenesis remains poorly understood.
  • The PIK3CA proto-oncogene is implicated in cell proliferation and survival in various cancers via the PI3K/Akt pathway.
  • Previous research identified PIK3CA somatic mutations and amplification in pituitary adenomas, but further investigation is warranted.

Purpose of the Study:

  • To investigate genetic alterations of the PIK3CA gene in a second series of pituitary adenomas.
  • To analyze PIK3CA mutations and amplification across different pituitary adenoma subtypes.

Main Methods:

  • Analysis of 33 pituitary adenomas of various subtypes (ACTH-secreting, GH-secreting, prolactin-secreting, nonfunctioning).
  • Direct sequencing of PIK3CA exons 9 and 20.
  • Quantitative PCR (qPCR) to assess genomic amplification (copy number ≥4).

Main Results:

  • PIK3CA mutations (exon 20) were detected in 12.1% (4/33) of cases.
  • PIK3CA amplification was observed in 21.2% (7/33) of cases.
  • A PIK3CA mutation was found in a noninvasive corticotroph microadenoma, contrasting with previous findings of mutations only in invasive tumors.

Conclusions:

  • This study confirms the presence of PIK3CA somatic mutations and amplifications in pituitary adenomas.
  • Findings support the involvement of the PI3K/Akt signaling pathway in pituitary tumorigenesis.
  • Further research into PIK3CA's role in different pituitary adenoma subtypes is indicated.

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