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Exogenous Administration of Microsomes-associated Alpha-synuclein Aggregates to Primary Neurons As a Powerful Cell Model of Fibrils Formation
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Exogenous Administration of Microsomes-associated Alpha-synuclein Aggregates to Primary Neurons As a Powerful Cell Model of Fibrils Formation

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An extracellular mechanism that can explain the neurotoxic effects of α-synuclein aggregates in the brain

C Pacheco1, L G Aguayo, C Opazo

  • 1Laboratory of Neurobiometals, Department of Physiology, University of Concepción Concepción, Chile.

Frontiers in Physiology
|August 31, 2012
PubMed

Keywords:
Parkinson's diseaseextracellular α-synuclein aggregatesneurodegenerationperforationplasma membranepore

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