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Related Experiment Video

Updated: May 18, 2026

Pulse-Wave Velocity, Flow-Mediated Dilation, and Carotid Intima-Media Thickness to Assess Cardiovascular Risk in Population with Metabolic Syndrome
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[Correlation between insulin resistance and endothelial dysfunction assessed by flow-mediated dilation].

Giuseppe Dattilo1, Annalisa Lamari, Sergio Crosca

  • 1UO di UTIC e Cardiologia, Ospedale di Urbino, Asur2 Marche, Urbino. giu.dattilo@libero.it

Recenti Progressi in Medicina
|October 2, 2012
PubMed
Summary

Insulin resistance is linked to endothelial dysfunction, a key cardiovascular risk factor. Ultrasound flow-mediated dilation (FMD) can detect these alterations, aiding in early risk assessment and therapy monitoring.

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Area of Science:

  • Cardiology
  • Endocrinology
  • Vascular Biology

Background:

  • Insulin resistance and endothelial dysfunction are significant contributors to cardiovascular disease.
  • Early identification of these risk factors is crucial for preventative strategies.

Purpose of the Study:

  • To investigate the association between insulin resistance and endothelial dysfunction.
  • To determine if endothelial function, assessed by flow-mediated dilation (FMD), is altered in patients with insulin resistance.

Main Methods:

  • A study population of 165 patients was assessed.
  • Thirty subjects with a family history of diabetes and/or hypoglycemia underwent evaluation of FMD, glucose/insulin curves, and HOMA index.
  • A subgroup of 20 patients with altered parameters was compared to 20 healthy controls.

Main Results:

  • A statistically significant difference was observed between the study and control groups in insulin curve performance, FMD measurements, and HOMA index.
  • The findings indicate a correlation between insulin resistance markers and impaired endothelial function.

Conclusions:

  • Flow-mediated dilation (FMD) is a valuable tool for assessing endothelial function in populations without apparent cardiovascular risk.
  • Monitoring FMD can help in evaluating the effectiveness of therapies aimed at protecting endothelial function.