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Emerging roles of Nrf2 and phase II antioxidant enzymes in neuroprotection
Meijuan Zhang1, Chengrui An, Yanqin Gao
1State Key Laboratory of Medical Neurobiology and Institute of Brain Science, Fudan University, Shanghai, China.
Abstract:
Phase II metabolic enzymes are a battery of critical proteins that detoxify xenobiotics by increasing their hydrophilicity and enhancing their disposal. These enzymes have long been studied for their preventative and protective effects against mutagens and carcinogens and for their regulation via the Keap1 (Kelch-like ECH associated protein 1)/Nrf2 (Nuclear factor erythroid 2 related factor 2)/ARE (antioxidant response elements) pathway. Recently, a series of studies have reported the altered expression of phase II genes in postmortem tissue of patients with various neurological diseases. These observations hint at a role for phase II enzymes in the evolution of such conditions. Furthermore, promising findings reveal that overexpression of phase II genes, either by genetic or chemical approaches, confers neuroprotection in vitro and in vivo. Therefore, there is a need to summarize the current literature on phase II genes in the central nervous system (CNS). This should help guide future studies on phase II genes as therapeutic targets in neurological diseases. In this review, we first briefly introduce the concept of phase I, II and III enzymes, with a special focus on phase II enzymes. We then discuss their expression regulation, their inducers and executors. Following this background, we expand our discussion to the neuroprotective effects of phase II enzymes and the potential application of Nrf2 inducers to the treatment of neurological diseases.
Insights
Phase II enzymes detoxify harmful substances and are regulated by the Keap1/Nrf2 pathway. Their altered expression in neurological diseases suggests potential as therapeutic targets for neuroprotection.
Area of Science:
- Biochemistry
- Neuroscience
- Toxicology
Background:
- Phase II metabolic enzymes detoxify xenobiotics, increasing hydrophilicity for excretion.
- These enzymes are crucial in preventing mutagen and carcinogen damage.
- The Keap1/Nrf2/ARE pathway regulates phase II enzyme expression.
Purpose of the Study:
- To review the role of phase II genes in the central nervous system (CNS).
- To explore the neuroprotective effects of phase II enzymes.
- To discuss the therapeutic potential of Nrf2 inducers for neurological diseases.
Main Methods:
- Literature review focusing on phase II enzymes and the CNS.
- Discussion of phase II enzyme expression, regulation, inducers, and executors.
- Analysis of studies on neuroprotection conferred by phase II gene overexpression.
Main Results:
- Altered expression of phase II genes is observed in postmortem CNS tissue of neurological disease patients.
- Overexpression of phase II genes demonstrates neuroprotective effects in vitro and in vivo.
- The Nrf2 pathway is a key regulator of these protective mechanisms.
Conclusions:
- Phase II enzymes play a significant role in the CNS and in the pathogenesis of neurological disorders.
- Targeting phase II genes, particularly via Nrf2 inducers, offers a promising therapeutic strategy for neurological diseases.
- Further research is warranted to fully elucidate and leverage the neuroprotective potential of phase II enzymes.
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