Are vesicles necessary for release of acetylcholine at cholinergic synapses?

Nitric oxide transforms serotonin into an inactive form and this affects neuromodulation.

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Calcium transients and neurotransmitter release at an identified synapse.

Trends in neurosciences·1999

Control of the calcium concentration involved in acetylcholine release and its facilitation: an additional role for synaptic vesicles?

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Cyclic ADP-ribose and calcium-induced calcium release regulate neurotransmitter release at a cholinergic synapse of Aplysia.

The Journal of physiology·1998

Opposite actions of nitric oxide on cholinergic synapses: which pathways?

Proceedings of the National Academy of Sciences of the United States of America·1996

NO decreases evoked quantal ACh release at a synapse of Aplysia by a mechanism independent of Ca2+ influx and protein kinase G.

The Journal of physiology·1996

Visceral adipose stem cell-derived small extracellular vesicles inhibit EMT in renal tubular epithelial cells and attenuate diabetic kidney fibrosis.

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The turmeric polyphenol calebin A relaxes rat aorta through CaV1.2 channel inhibition with evidence for KCa1.1 channel involvement.

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DNMT1 is associated with anti-PD-1 resistance and regulates the STAT3/PD-L1-CD8+ T-cell axis in melanoma.

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Biochemical pharmacology·2026

Neuronal activity-driven 3D chromatin dynamics in cortical pyramidal neurons depend on SATB2.

Nucleic acids research·2026

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Frontiers in molecular neuroscience·2026

Early-life oxidative stress programs persistent NFκB activation and neuroinflammation in autism spectrum disorder.

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Journal of Nepal Health Research Council·2026

NADPH oxidases in immunometabolism and disease pathology: mechanistic networks, pollutant triggers, and therapeutic frontiers.

Cellular & molecular immunology·2026

Structural insights enable drug discovery for the neuronal NBCn2 carbonate transporter.

Nature communications·2026

Related Experiment Videos

Are vesicles necessary for release of acetylcholine at cholinergic synapses?

Related Concept Videos

Nitric oxide transforms serotonin into an inactive form and this affects neuromodulation.

Calcium transients and neurotransmitter release at an identified synapse.

Control of the calcium concentration involved in acetylcholine release and its facilitation: an additional role for synaptic vesicles?

Cyclic ADP-ribose and calcium-induced calcium release regulate neurotransmitter release at a cholinergic synapse of Aplysia.

Opposite actions of nitric oxide on cholinergic synapses: which pathways?

NO decreases evoked quantal ACh release at a synapse of Aplysia by a mechanism independent of Ca2+ influx and protein kinase G.

Visceral adipose stem cell-derived small extracellular vesicles inhibit EMT in renal tubular epithelial cells and attenuate diabetic kidney fibrosis.

From intercellular communication to therapeutic delivery: Mechanistic perspectives of cancer exosomes.

A novel stilbene derivative targets RAD51 activity and sensitizes DU145 prostate cancer cells to cisplatin.

The turmeric polyphenol calebin A relaxes rat aorta through CaV1.2 channel inhibition with evidence for KCa1.1 channel involvement.

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Are vesicles necessary for release of acetylcholine at cholinergic synapses?

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