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Related Concept Videos

Pleiotropy01:33

Pleiotropy

Pleiotropy is the phenomenon in which a single gene impacts multiple, seemingly unrelated phenotypic traits. For example, defects in the SOX10 gene cause Waardenburg Syndrome Type 4, or WS4, which can cause defects in pigmentation, hearing impairments, and an absence of intestinal contractions necessary for elimination. This diversity of phenotypes results from the expression pattern of SOX10 in early embryonic and fetal development. SOX10 is found in neural crest cells that form melanocytes,...
Nephrotic Syndrome I : Introduction01:24

Nephrotic Syndrome I : Introduction

Nephrotic Syndrome is a chronic kidney disorder defined by clinical findings such as severe proteinuria, hypoalbuminemia, hyperlipidemia, and edema. These symptoms result from damage to the glomeruli, the kidney’s filtering units, increasing their permeability to proteins.Definition and Meaning:Proteinuria, defined as the loss of more than 3.5 grams of protein per day in adults, is a crucial feature of nephrotic syndrome. This condition is often accompanied by edema, the accumulation of fluid...
Disorder of Water Balance01:29

Disorder of Water Balance

Water balance disorders are medical conditions that occur when there is a deviation from the body's water volume or osmolarity, disrupting normal homeostasis and leading todehydration, hypotonic hydration, hyperhydration, edema, or water intoxication.
Dehydration
Dehydration occurs when the body loses fluids (particularly water).
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Renal Tubule and Collecting Duct01:24

Renal Tubule and Collecting Duct

The renal tubule is divided into three parts: the proximal convoluted tubule (PCT), the Loop of Henle (LOH), and the distal convoluted tubule (DCT).
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Nephrotic Syndrome II : Assessment and Medical Management01:26

Nephrotic Syndrome II : Assessment and Medical Management

IntroductionNephrotic syndrome is a kidney disorder marked by excessive protein loss in the urine, leading to various systemic complications. This condition often results from damage to the glomeruli—the kidney's filtering units—causing proteinuria, low blood protein levels, and fluid retention. Understanding the assessment, diagnosis, and management of nephrotic syndrome is essential for effective treatment and prevention of further kidney damage.AssessmentPatient History: Document any history...
Cushing Syndrome II: Pathophysiology01:19

Cushing Syndrome II: Pathophysiology

Cortisol production is normally governed by the hypothalamic–pituitary–adrenal (HPA) axis, which maintains hormonal balance through tightly regulated feedback mechanisms. Disruption of this regulatory system is central to the development of Cushing syndrome, whether the excess cortisol originates from external medications or internal pathology. Persistent cortisol elevation alters metabolism, immune function, and endocrine signaling, producing the characteristic clinical features of the...

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Isolation of Neonatal Extrahepatic Cholangiocytes
07:54

Isolation of Neonatal Extrahepatic Cholangiocytes

Published on: June 5, 2014

Wells syndrome.

Manish Bansal1, Tulika Rai, Shyam S Pandey

  • 1Department of Dermatology and Venereology, Institute of Medical Sciences, Banaras Hindu University, Varanasi, Uttar Pradesh, India.

Indian Dermatology Online Journal
|November 29, 2012
PubMed
Summary
This summary is machine-generated.

Wells syndrome, also known as eosinophilic cellulitis, presents as skin inflammation. This case highlights a rare presentation of recurrent itchy plaques on the forearm in a 32-year-old female.

Keywords:
CellulitisWells syndromeeosinophils

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Area of Science:

  • Dermatology
  • Pathology

Background:

  • Wells syndrome (eosinophilic cellulitis) is an inflammatory skin condition.
  • It is characterized by clinical presentation resembling cellulitis and histopathological findings of eosinophilic infiltration in the dermis.
  • Diverse clinical presentations of Wells syndrome have been documented.

Observation:

  • A 32-year-old female presented with an 8-month history of recurrent, erythematous plaques on her left forearm.
  • The plaques were associated with mild itching.
  • The lesions resolved, leaving behind mild hyperpigmentation.

Findings:

  • The case describes a unique morphological presentation of Wells syndrome.
  • Recurrent erythematous plaques with itching and subsequent hyperpigmentation are key features.

Implications:

  • This case expands the spectrum of clinical presentations for Wells syndrome.
  • Recognizing diverse presentations is crucial for accurate diagnosis and management of eosinophilic cellulitis.