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The Joint Effect of Social Comparison and Social Distance on Evaluation of Intertemporal Choice Outcomes in Event-related Potential Studies

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A NET Outcome.

Thea Lu¹, Scott D Kobayashi, Mark T Quinn

¹Laboratory of Human Bacterial Pathogenesis, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, National Institutes of Health Hamilton, MT, USA.

Frontiers in Immunology

|December 11, 2012

Summary

This summary is machine-generated.

Neutrophils release neutrophil extracellular traps (NETs) to kill microbes via a process called NETosis. This review discusses the mechanisms and functions of NETs in innate immunity.

Keywords:

apoptosis inflammation necrosis neutrophil phagocytosis

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Area of Science:

Immunology
Cell Biology

Background:

Neutrophils are key immune cells in innate immunity, primarily known for phagocytosis.
Neutrophil Extracellular Traps (NETs) represent a non-phagocytic antimicrobial mechanism.

Purpose of the Study:

To review and provide perspective on the formation, mechanisms, and functions of NETs and NETosis.
To address unanswered questions regarding NETs in host defense.

Main Methods:

Literature review and synthesis of existing research on NETs and NETosis.
Discussion of in vitro findings and their implications for in vivo functions.

Main Results:

NETs are composed of DNA, histones, and antimicrobial proteins.
NETosis is a distinct cell death pathway involved in NET formation.
The precise molecular mechanisms and in vivo roles of NETs require further elucidation.

Conclusions:

NETs are a significant component of the innate immune response.
Further research is needed to fully understand NETosis and the therapeutic potential of targeting NETs.