Cholesterol accumulation in the arterial cells and in the extracellular spaces

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Lamellar body membrane turnover is stimulated by secretagogues.

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Mechanism for secretagogue-induced surfactant protein A binding to lung epithelial cells.

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Macrophages primed by overnight culture demonstrate a marked stimulation of surfactant protein A degradation.

The American journal of physiology·1997

Bromism: intoxication from a rare anticonvulsant therapy.

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Exclusion probabilities of 22 bovine microsatellite markers in fluorescent multiplexes for semiautomated parentage testing.

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Aldehyde induced hypertension in rats: prevention by N-acetyl cysteine.

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Effects of a nitric oxide synthase inhibitor on pressor response to angiotensin II in conscious rats.

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Vascular innervation in atherogenesis.

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Leukocyte flow properties, polymorphonuclear membrane fluidity and cytosolic Ca2+ content in subjects with vascular atherosclerotic disease.

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Cholesterol accumulation in the arterial cells and in the extracellular spaces

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Surface-expressed lamellar body membrane is recycled to lamellar bodies.

Lamellar body membrane turnover is stimulated by secretagogues.

Mechanism for secretagogue-induced surfactant protein A binding to lung epithelial cells.

Macrophages primed by overnight culture demonstrate a marked stimulation of surfactant protein A degradation.

Bromism: intoxication from a rare anticonvulsant therapy.

Exclusion probabilities of 22 bovine microsatellite markers in fluorescent multiplexes for semiautomated parentage testing.

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