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Internalization of human immunodeficiency virus type I and other retroviruses by megakaryocytes and platelets

D Zucker-Franklin1, S Seremetis, Z Y Zheng

  • 1Department of Medicine, New York University Medical Center, NY.

Blood
|May 15, 1990
PubMed

Insights

Human immunodeficiency virus type I (HIV-I) can directly infect megakaryocytes and platelets. This finding suggests a potential role for direct viral infection in HIV-associated disease and identifies megakaryocytes as potential targets for gene therapy.

Area of Science:

  • Virology
  • Hematology
  • Cell Biology

Background:

  • Direct infection of megakaryocytes and platelets by human immunodeficiency virus type I (HIV-I) or other retroviruses has not been previously demonstrated.
  • Understanding the cellular targets of HIV-I is crucial for comprehending viral pathogenesis and developing effective therapies.

Purpose of the Study:

  • To investigate the potential for direct infection of megakaryocytes and platelets by retroviruses, including HIV-I.
  • To explore the implications of megakaryocyte and platelet susceptibility for HIV-I pathophysiology and gene transfer applications.

Main Methods:

  • Co-cultivation of murine bone marrow with a retrovirus-producing cell line (PA317-N2).
  • Co-cultivation of human bone marrow and platelets with HIV-infected H9 cells.
  • Ultrastructural analysis to detect viral presence within megakaryocytes and platelets.

Main Results:

  • Ultrastructural analyses confirmed the presence of viruses within megakaryocytes and platelets in both murine and human specimens.
  • Murine megakaryocyte differentiation stages showed avid retroviral uptake.
  • Human specimens exhibited less frequent, but detectable, viral uptake by megakaryocytes and platelets.

Conclusions:

  • Direct infection of megakaryocytes by HIV-I is possible, suggesting a role in HIV-associated disease.
  • Megakaryocyte lineage cells may serve as target cells for future retroviral-based gene transfer strategies.

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