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Endovascular interventions for TransAtlantic InterSociety Consensus II C and D femoropopliteal lesions

Min-yi Yin1, Mi-er Jiang, Xin-tian Huang

  • 1Department of Vascular Surgery, Shanghai 9th People's Hospital, Shanghai Jiao Tong University, Shanghai 200011, China.

Chinese Medical Journal
|February 21, 2013
PubMed

Insights

Endovascular treatment for severe femoropopliteal artery disease (TASC II C/D) shows high success and good patency rates. Key factors for restenosis include hyperlipidemia, lesion length, and popliteal artery involvement.

Area of Science:

  • Vascular Surgery
  • Interventional Cardiology
  • Endovascular Interventions

Background:

  • Peripheral artery disease (PAD) causes significant morbidity and hospitalizations.
  • Endovascular techniques offer an aggressive approach to PAD treatment.
  • TASC II C and D lesions represent complex femoropopliteal occlusive disease.

Purpose of the Study:

  • To evaluate outcomes of endovascular interventions for TASC II C and D femoropopliteal occlusive disease.
  • To determine technical success, complication rates, and patency.
  • To identify predictors of restenosis/occlusion.

Main Methods:

  • Retrospective review of patients undergoing endovascular intervention for femoropopliteal occlusive disease (2007-2010).
  • Data collected: demographics, risk factors, ABI, technical success, complications.
  • Patency assessed by Kaplan-Meier analysis; univariate/multivariate analyses for predictors.

Main Results:

  • 91.1% technical success rate; 12.0% complication rate (5.1% major).
  • Primary patency at 1 year: 95% (TASC II C) vs. 89% (TASC II D).
  • Secondary patency at 4 years: 94% (TASC II C) vs. 83% (TASC II D). Hyperlipidemia, lesion length, popliteal involvement predicted restenosis.

Conclusions:

  • Endovascular treatment of TASC II C/D femoropopliteal disease achieves high technical success.
  • Favorable mid-term secondary patency rates were observed.
  • Hyperlipidemia, lesion length, and popliteal artery involvement are independent risk factors for in-stent restenosis.
Abstract

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