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Related Concept Videos

Portal Hypertension01:22

Portal Hypertension

Portal hypertension is an increase in blood pressure within the portal venous system. Normally, this pressure is less than 5 mmHg. It is considered clinically significant when it rises above 10 mmHg. At this threshold, complications from altered blood flow and venous congestion emerge.EtiologyPortal hypertension arises from conditions that impede blood flow through the liver. The most common cause is cirrhosis, in which chronic liver injury leads to fibrotic scarring. This fibrosis narrows or...
Pulmonary Hypertension: Classification and Pathogenesis01:30

Pulmonary Hypertension: Classification and Pathogenesis

Pulmonary hypertension (PH) is a severe health condition in which the mean pulmonary arterial pressure increases to 25 mmHg or more, even when the body is at rest. This high pressure in the blood vessels that transport blood from the heart to the lungs can cause various symptoms, including shortness of breath, can lead to right heart failure, and significantly affect the overall quality of life.
There are various classifications for PH, each relating to different underlying causes and also...
Peptic Ulcer01:27

Peptic Ulcer

Peptic ulcers are erosive lesions of the gastric or duodenal lining, most commonly caused by Helicobacter pylori infection. This Gram-negative, helical bacterium has adapted to survive the stomach’s acidic environment by producing urease, which converts urea into ammonia and carbon dioxide. The ammonia neutralizes gastric acid in the bacterium’s immediate environment, allowing colonization of the gastric mucosa. H. pylori attaches to mucus-secreting epithelial cells, penetrates the mucus...
Hypertension III: Clinical Manifestations and Diagnostic Studies01:30

Hypertension III: Clinical Manifestations and Diagnostic Studies

Hypertension is asymptomatic and also referred to as the "silent killer" until it progresses to a severe stage or causes target organ disease. Patients may experience symptoms stemming from the strain on blood vessels and tissues in various organs or the heart's increased workload.Physical exams might show no abnormalities other than high blood pressure. Signs of vascular damage, when present, correspond to the organs supplied by the affected vessels, leading to target organ damage. For...
Peptic Ulcer Disease III: Clinical Manifestations and Diagnostic Studies01:28

Peptic Ulcer Disease III: Clinical Manifestations and Diagnostic Studies

Peptic ulcer disease (PUD) presents with diverse symptoms depending on the location and severity of the ulcer. Clinical manifestations of peptic ulcer include dull pain and a burning sensation in the mid-epigastric region.
Few clinical manifestations differentiate gastric ulcers from duodenal ulcers. Distinctions in the location, timing, and pain relief are crucial for healthcare providers in differentiating between gastric and duodenal ulcers during clinical assessments.
Diverticular Disease of the Colon01:27

Diverticular Disease of the Colon

Diverticular disease involves the formation of diverticula—small sac-like outpouchings of the colonic wall—and their complications. It most commonly affects the sigmoid colon due to higher intraluminal pressure and structural vulnerability. It results from structural weakness and increased pressure in the colon, producing pseudodiverticula that may remain silent or progress to inflammation and serious complications.Structure of DiverticulaIn diverticulosis, these outpouchings are...

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Updated: May 13, 2026

Invasive Hemodynamic Characterization of the Portal-hypertensive Syndrome in Cirrhotic Rats
09:37

Invasive Hemodynamic Characterization of the Portal-hypertensive Syndrome in Cirrhotic Rats

Published on: August 1, 2018

Portal hypertensive polyps: distinct entity.

Anjali D Amarapurkar1, Deepak Amarapurkar, Mehul Choksi

  • 1Department Pathology, SRL and Dr Avinash Phadke Laboratory, Mumbai 400 008, India. anjali1963@gmail.com

Indian Journal of Gastroenterology : Official Journal of the Indian Society of Gastroenterology
|March 21, 2013
PubMed
Summary
This summary is machine-generated.

Portal hypertensive polyps (PHPs) are identifiable lesions in patients with portal hypertension (PH). These polyps are likely linked to increased angiogenesis in the gastric mucosa.

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Area of Science:

  • Gastroenterology
  • Pathology
  • Angiogenesis Research

Background:

  • Gastric mucosal alterations in portal hypertension (PH) are recognized.
  • Gastroduodenal polyps in PH are infrequently documented.

Purpose of the Study:

  • To determine the prevalence of upper gastrointestinal (GI) polyps in patients with portal hypertension (PH) from any cause.
  • To investigate the role of angiogenesis in the development of portal hypertensive polyps (PHPs).

Main Methods:

  • Retrospective analysis of upper GI endoscopies to compare polyp etiology in PH versus non-PH groups.
  • Histological confirmation of polyp diagnoses.
  • Prospective analysis of CD 34 expression and morphometry in 47 patients.

Main Results:

  • Out of 3,811 endoscopies, 121 patients (3.2%) had polyps. In 631 PH patients, 16 had polyps (9 PHPs).
  • Polyp prevalence was similar between PH (2.5%) and non-PH (3.3%) groups.
  • PH polyps and gastric mucosa showed increased vessel diameter (>50 μm) and density compared to controls.

Conclusions:

  • Portal hypertensive polyps (PHPs) are distinct lesions in patients with cirrhosis and PH.
  • PHPs are associated with increased angiogenesis in the gastric mucosa.