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Related Concept Videos

Peripheral Artery Disease I: Introduction01:30

Peripheral Artery Disease I: Introduction

Peripheral artery disease (PAD) predominantly results from atherosclerosis, which involves the accumulation of fatty deposits, or plaques, within the walls of arteries. This causes them to narrow and harden, significantly reducing blood flow. PAD predominantly affects the legs, particularly the arteries supplying the thighs and calves. In rare cases, it may involve other arteries, including those in the arms.Etiology of PAD:The principal cause of PAD is atherosclerosis, which results from fatty...
Peripheral Arterial Disease II: Clinical Manifestations and Diagnostic Evaluation01:21

Peripheral Arterial Disease II: Clinical Manifestations and Diagnostic Evaluation

Clinical manifestationsPeripheral Arterial Disease (PAD) manifests through a range of symptoms, from the characteristic intermittent claudication to atypical presentations and severe complications in advanced stages. Intermittent claudication, a hallmark symptom of PAD, presents as exercise-induced muscle pain that typically resolves within minutes of rest. This pain is reproducible and stems from inadequate blood flow, leading to the accumulation of lactic acid produced during anaerobic...
Nephrotic Syndrome I : Introduction01:24

Nephrotic Syndrome I : Introduction

Nephrotic Syndrome is a chronic kidney disorder defined by clinical findings such as severe proteinuria, hypoalbuminemia, hyperlipidemia, and edema. These symptoms result from damage to the glomeruli, the kidney’s filtering units, increasing their permeability to proteins.Definition and Meaning:Proteinuria, defined as the loss of more than 3.5 grams of protein per day in adults, is a crucial feature of nephrotic syndrome. This condition is often accompanied by edema, the accumulation of fluid...
Peripheral Artery Disease III: Interprofessional Care01:27

Peripheral Artery Disease III: Interprofessional Care

Peripheral Artery Disease (PAD) is characterized by narrowed arteries that diminish blood flow to the extremities. Effective management of PAD requires an interprofessional approach involving various healthcare professionals. The critical aspects of interprofessional care for PAD patients focus on risk factor modification, drug therapy, exercise therapy, nutrition therapy, critical limb ischemia care, and interventional radiology and surgical procedures.The primary treatment goal for PAD...
Nephrotic Syndrome II : Assessment and Medical Management01:26

Nephrotic Syndrome II : Assessment and Medical Management

IntroductionNephrotic syndrome is a kidney disorder marked by excessive protein loss in the urine, leading to various systemic complications. This condition often results from damage to the glomeruli—the kidney's filtering units—causing proteinuria, low blood protein levels, and fluid retention. Understanding the assessment, diagnosis, and management of nephrotic syndrome is essential for effective treatment and prevention of further kidney damage.AssessmentPatient History: Document any history...
Peripheral Artery Disease IV: Nursing Management01:26

Peripheral Artery Disease IV: Nursing Management

The nursing management of a patient with peripheral artery disease (PAD) begins with a thorough assessment of the patient’s health history and clinical manifestations.AssessmentHealth History: Evaluate the patient’s history of hypertension, hyperlipidemia, family history of cardiovascular issues, and lifestyle factors such as dietary patterns, smoking, and physical activity.Physical Examination:Assess the affected extremity for decreased or absent peripheral pulses, temperature changes,...

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Mechanism of Kemeng Fang's Inhibition of Podocyte Apoptosis in Rats with Membranous Nephropathy through the PI3K/AKT Signaling Pathway
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Mechanism of Kemeng Fang's Inhibition of Podocyte Apoptosis in Rats with Membranous Nephropathy through the PI3K/AKT Signaling Pathway

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Cutaneous polyarteritis nodosa: an update.

Fukumi Furukawa1

  • 1Department of Dermatology, Wakayama Medical University, Wakayama, Wakayama, Japan.

Annals of Vascular Diseases
|April 5, 2013
PubMed
Summary

Cutaneous polyarteritis nodosa (CPN) is a skin-limited form of polyarteritis nodosa (PN) with a benign prognosis. This study suggests CPN does not progress to PN and proposes new diagnostic criteria for both conditions.

Keywords:
clinical entitycutaneous polyarteritis nodosadefinitiondiagnostic criteriaextracutaneous symptoms

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Granulocyte-dependent Autoantibody-induced Skin Blistering
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Granulocyte-dependent Autoantibody-induced Skin Blistering

Published on: October 12, 2012

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Mechanism of Kemeng Fang's Inhibition of Podocyte Apoptosis in Rats with Membranous Nephropathy through the PI3K/AKT Signaling Pathway
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Published on: August 23, 2024

Granulocyte-dependent Autoantibody-induced Skin Blistering
12:23

Granulocyte-dependent Autoantibody-induced Skin Blistering

Published on: October 12, 2012

Area of Science:

  • Dermatology
  • Rheumatology
  • Vasculitis Research

Background:

  • Cutaneous symptoms are common in polyarteritis nodosa (PN), with cutaneous polyarteritis nodosa (CPN) representing a skin-limited form.
  • Debate exists on whether CPN can progress to systemic PN, despite its generally benign prognosis.
  • Current diagnostic criteria for PN and CPN lack specificity, necessitating clearer definitions.

Purpose of the Study:

  • To redefine the clinical entity of CPN.
  • To propose appropriate diagnostic criteria for CPN and PN.
  • To investigate the progression of CPN to systemic PN.

Main Methods:

  • Retrospective collection of 22 CPN cases with histopathological confirmation from Japanese clinics.
  • Analysis of clinical and histopathological manifestations, including extracutaneous symptoms.
  • Consultation with 17 dermatological specialists in vasculitis.

Main Results:

  • None of the 22 CPN cases progressed to PN or death during follow-up.
  • Peripheral neuropathy was observed in 32% of cases, and myalgia in 27%.
  • Extracutaneous symptoms in CPN were considered limited to the affected dermatomes.

Conclusions:

  • CPN is suggested to be a distinct entity that does not progress to systemic PN.
  • New diagnostic criteria for CPN and PN were proposed and recognized as appropriate by dermatologists.
  • The study emphasizes the importance of clear diagnostic criteria for differentiating CPN and PN.