Related Experiment Video
Updated: May 12, 2026
06:26
Novel and Innovative Hybrid Technique for Type A Aortic Dissection
Published on: March 28, 2025
Acute ischemic pancreatitis associated with acute type B aortic dissection: a case report
1Department of Cardiovascular Surgery, Onomichi General Hospital, Onomichi, Hiroshima, Japan.
Annals of Vascular Diseases
|April 5, 2013
Summary
A man with acute type B aortic dissection developed pancreatitis. Conservative management resolved both conditions, avoiding surgery.
Area of Science:
- Cardiology
- Vascular Surgery
- Gastroenterology
Background:
- Acute type B aortic dissection (ATB-AD) is a life-threatening condition requiring prompt diagnosis and management.
- Visceral artery involvement in ATB-AD can lead to ischemic complications.
- Pancreatitis is a potential, though less common, complication of ATB-AD.
Observation:
- A 47-year-old male presented with sudden back pain, diagnosed with ATB-AD extending to celiac and splenic arteries.
- Following antihypertensive treatment, the patient developed upper abdominal pain and elevated amylase levels.
- Computed tomography (CT) revealed peripancreatic fluid, indicating acute pancreatitis, without further aortic or visceral dissection.
Findings:
- Conservative management with antihypertensive therapy and protease inhibitors for pancreatitis was initiated.
- Follow-up CT demonstrated resolution of the thrombosed splenic artery false lumen and reduced peripancreatic effusion.
- The patient's pancreatitis and aortic dissection symptoms resolved, leading to discharge without surgical intervention.
Implications:
- This case highlights the importance of monitoring for abdominal complications in patients with ATB-AD.
- Non-surgical management can be effective for managing pancreatitis secondary to aortic dissection.
- Early recognition and conservative treatment strategies are crucial for favorable outcomes in complex aortic dissections.
Related Concept Videos
01:25Acute Pancreatitis I: Introduction
Acute pancreatitis is the sudden inflammation of the pancreas caused by the early activation of digestive enzymes, leading to the autodigestion of pancreatic tissue. This results in local inflammation and, in severe cases, systemic complications.EtiologyUnderstanding the underlying causes is crucial, as identifying the etiology guides treatment and anticipates complications. Acute pancreatitis can be triggered by various factors, typically grouped into the following clinical categories.Biliary...
01:27Acute Pancreatitis I: Introduction
Pancreatitis is inflammation of the pancreas, an organ located behind the stomach. It can be either acute or chronic.
Acute pancreatitis is characterized by rapid inflammation of the pancreas, often caused by factors like gallstone blockage or excessive alcohol consumption. Chronic pancreatitis, on the other hand, is a slow, progressive inflammation that may result from long-term alcohol abuse, obstructions in the pancreatic duct, or genetic factors.
The causes of acute pancreatitis include:
Acute pancreatitis is characterized by rapid inflammation of the pancreas, often caused by factors like gallstone blockage or excessive alcohol consumption. Chronic pancreatitis, on the other hand, is a slow, progressive inflammation that may result from long-term alcohol abuse, obstructions in the pancreatic duct, or genetic factors.
The causes of acute pancreatitis include:
01:30Acute Pancreatitis II: Clinical Manifestations and Management
Acute pancreatitis presents a complex medical emergency characterized by rapid onset inflammation of the pancreas, demanding timely diagnosis and management to prevent complications. The condition primarily manifests through severe upper abdominal pain that often radiates to the back. This pain intensifies following the consumption of fatty foods. Accompanying symptoms such as nausea, vomiting, abdominal distention, fever, dyspnea, cyanosis, and jaundice can vary in intensity but significantly...
01:21Acute Pancreatitis II: Pathophysiology
The pathophysiology of acute pancreatitis centers on injury to pancreatic acinar cells, which initiates a cascade of harmful intracellular events.This injury leads to premature activation of trypsinogen to trypsin in the pancreas. Trypsin then activates other digestive enzymes, such as chymotrypsin, elastase, and phospholipase A2, which begin breaking down pancreatic tissue. The resulting autodigestion causes local inflammation, tissue swelling, hemorrhage, and fat necrosis.Injured acinar cells...
01:21Aneurysm II: Clinical Manifestations and Diagnostic Studies
Thoracic, aortic arch and abdominal aneurysms are significant vascular conditions that can present with various clinical manifestations and lead to serious complications. Understanding these manifestations and the appropriate diagnostic studies is essential for effective management and treatment.Thoracic Aortic AneurysmsThoracic aortic aneurysms often remain asymptomatic until they reach a size that impinges on adjacent structures. They typically cause deep, diffuse chest pain that radiates to...
01:26Aneurysm III: Interprofessional Care
Aneurysm management involves either conservative medical therapy or surgical intervention, depending on the size and symptoms of the aneurysm. Conservative management is generally reserved for smaller, asymptomatic aneurysms, while larger or symptomatic aneurysms often necessitate surgical repair.Conservative Medical TherapyFor small, asymptomatic aneurysms, particularly abdominal aortic aneurysms (AAA) less than 5.5 centimeters in diameter, conservative medical therapy is recommended. This...