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Tonsillitis II: Management01:26

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Chronic Pharyngitis01:23

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Chronic pharyngitis refers to persistent inflammation of the pharyngial mucosa.
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Chronic pancreatitis is a progressive and irreversible inflammation of the pancreas, most often caused by long-term alcohol abuse, but it can also be related to ductal obstruction, smoking, or genetic factors.Chronic pancreatitis occurs when the pancreas is repeatedly exposed to harmful agents like alcohol, smoking, ductal obstruction, or genetic predisposition. These factors lead to the release of toxic metabolites and inflammatory cytokines, sustaining chronic inflammation in the pancreatic...
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Updated: May 12, 2026

Studying Effects of Cigarette Smoke on Pseudomonas Infection in Lung Epithelial Cells
09:15

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Published on: May 11, 2020

Smoking promotes peritonsillar abscess.

Tejs Ehlers Klug1, Maria Rusan, Kim Katrine Bjerring Clemmensen

  • 1Department of Otorhinolaryngology, Head and Neck Surgery, Aarhus University Hospital, NBG, Building 10, Noerrebrogade 44, Aarhus C, 8000, Denmark, tejsehlersklug@hotmail.com.

European Archives of Oto-Rhino-Laryngology : Official Journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : Affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery
|April 10, 2013
PubMed
Summary

Smoking significantly increases the risk of developing peritonsillar abscess (PTA), a complication of acute tonsillitis. This study found no association between smoking and specific bacteria in PTA cases.

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Area of Science:

  • Otolaryngology
  • Epidemiology
  • Public Health

Background:

  • Peritonsillar abscess (PTA) frequently complicates acute tonsillitis, particularly in adolescents and young adults.
  • Smoking initiation commonly occurs during adolescence and young adulthood, suggesting a potential link to PTA.

Purpose of the Study:

  • To investigate if smoking elevates the risk of developing PTA.
  • To determine if smoking is associated with specific bacterial findings in PTA patients.

Main Methods:

  • Retrospective study of PTA patients admitted to Aarhus University Hospital (2001-2006).
  • Analysis of smoking habits, age, and gender data, comparing PTA patients to the general population.
  • Microbiological analysis of PTA samples.

Main Results:

  • Smoking was associated with a significantly increased risk of PTA (odds ratios 1.9-4.7).
  • Higher PTA incidence in males compared to females was partly explained by higher male smoking prevalence.
  • No significant differences in bacterial findings (Fusobacterium necrophorum, beta-hemolytic streptococci) between smokers and non-smokers with PTA were observed.

Conclusions:

  • Smoking is a significant risk factor for PTA across all ages and genders.
  • While smoking increases PTA risk, it does not appear to alter the associated bacterial flora.