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A case of supracondylar humerus fracture with vascular impairment. Open it up?
Stefanie Erhart1, M Lutz, P Larcher
1Department for Trauma Surgery, Medical University Innsbruck, Anichstraße 35, 6020, Innsbruck, Austria. stefanie.erhart@uki.at
Insights
Pediatric supracondylar humerus fractures can cause serious neurovascular injury. Prompt surgical exploration and brachial artery repair with a vein graft ensured full hand function in a 3-year-old girl.
Area of Science:
- Pediatric Orthopedics
- Vascular Surgery
- Emergency Medicine
Background:
- Supracondylar humerus fractures are common in children.
- Neurovascular compromise is a significant complication, potentially leading to severe functional impairment.
Observation:
- A 3-year-old girl presented with a supracondylar humerus fracture.
- Initially well-perfused, her hand became pulseless during surgical preparation.
- This highlighted the challenge in diagnosing vascular injury versus spontaneous pulse loss.
Findings:
- Open reduction and internal pin fixation were performed.
- Surgical exploration revealed brachial artery injury.
- Successful arterial repair was achieved using a cephalic vein graft.
Implications:
- This case underscores the importance of frequent clinical reevaluation in pediatric supracondylar humerus fractures.
- Early surgical intervention and vascular repair are crucial for preserving limb function.
- Differentiating true vascular injury from transient pulselessness requires vigilance and timely surgical assessment.
Abstract:
Supracondylar humerus fracture is one of the most frequent fractures in childhood. A serious complication is an injury to the neurovascular structures which could potentially result in severe functional impairment. We are presenting the case of a 3-year-old girl with a supracondylar humerus fracture in our emergency department and highlight the diagnostic and therapeutic steps in managing this situation. Initially, the hand was well perfused but showed to be pulseless in the operating theater after preparation for surgery. After open reduction and internal pin fixation the neurovascular structures were explored and the brachial artery was repaired by means of a cephalic vein graft. On follow up the patient presented with normal hand function and without vascular or neurologic deficits. As concomitant vascular injuries after supracondylar humerus fractures are rare, it can be difficult to discriminate a pink pulseless hand from a patient with essential vascular injury. Our case also demonstrates the need for short-term reevaluation of the clinical status. When in doubt there should be no hesitation to perform open surgery and vascular repair.
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