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Semi-quantitative Assessment Using [18F]FDG Tracer in Patients with Severe Brain Injury
Published on: November 9, 2018
Single case study of brain FDG PET imaging in a patient with catatonia
Dane Breker1, Nicolaas I Bohnen
1Department of Neurology, University of Michigan, Taubman 1st Floor, Reception C, 1500 E. Medical Center Drive, SPC 5316, Ann Arbor, Michigan 48109, USA. dbreker@med.umich.edu
Abstract:
We report a case of bilateral occipitotemporal and thalamic hypometabolism on FDG PET in a 19-year-old male patient who presented with altered mental status. He had a history of static encephalopathy and presented with visual hallucinations and decreased verbal output, sleep, and appetite 7 days after starting amoxicillin for otitis media. Extensive evaluation failed to demonstrate any infectious, inflammatory, autoimmune-mediated (including negative testing for NMDA receptor antibody), paraneoplastic, or toxic-metabolic etiology. Ultimately, he responded well to a benzodiazepine challenge and electroconvulsive therapy and was diagnosed with catatonia.
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